pubmed-article:6782204 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0005767 | lld:lifeskim |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0086045 | lld:lifeskim |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0013618 | lld:lifeskim |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0023175 | lld:lifeskim |
pubmed-article:6782204 | lifeskim:mentions | umls-concept:C0005884 | lld:lifeskim |
pubmed-article:6782204 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:6782204 | pubmed:dateCreated | 1981-5-13 | lld:pubmed |
pubmed-article:6782204 | pubmed:abstractText | The cheletable part of lead body burden was measured in 32 workers and seven office workers after an infusion test with CaNa2EDTA. The workers had been exposed to lead at a lead and zinc processing unit for one to three years (mean one year). There was good correlation (r = 0.87) between blood lead and chelatable urinary lead excretion described by the equation y = 0.07 . 10(0.46.x). From this equation it can be predicted that the generally accepted limit value for chelatable urinary lead excretion, 0.42 mumol/mmol CaNa2EDTA administered per 24 hours (3.1 mumol/24 hours or 650 micrograms/24 hours), corresponds to a blood lead concentration (PbB) of 1.7 mumol/l (or 35 micrograms/100 ml), which is lower than the commonly accepted limit value of 2.9 mumol/l (or 60 micrograms/100 ml) for occupationally lead-exposed persons. There was a better correlation between the cheletable lead excretion and the urinary ALA-excretion (r = 0.45; p less than 0.001) than between PbB and the urinary ALA-excretion (r = 0.26; p greater than 0.05). | lld:pubmed |
pubmed-article:6782204 | pubmed:language | eng | lld:pubmed |
pubmed-article:6782204 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6782204 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:6782204 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:6782204 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6782204 | pubmed:month | Jan | lld:pubmed |
pubmed-article:6782204 | pubmed:issn | 0096-1736 | lld:pubmed |
pubmed-article:6782204 | pubmed:author | pubmed-author:DøssingMM | lld:pubmed |
pubmed-article:6782204 | pubmed:author | pubmed-author:HansenJ PJP | lld:pubmed |
pubmed-article:6782204 | pubmed:author | pubmed-author:PaulevP EPE | lld:pubmed |
pubmed-article:6782204 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6782204 | pubmed:volume | 23 | lld:pubmed |
pubmed-article:6782204 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6782204 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6782204 | pubmed:pagination | 39-43 | lld:pubmed |
pubmed-article:6782204 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
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pubmed-article:6782204 | pubmed:meshHeading | pubmed-meshheading:6782204-... | lld:pubmed |
pubmed-article:6782204 | pubmed:year | 1981 | lld:pubmed |
pubmed-article:6782204 | pubmed:articleTitle | Chelatable lead body burden (by calcium-disodium EDTA) and blood lead concentration in man. | lld:pubmed |
pubmed-article:6782204 | pubmed:publicationType | Journal Article | lld:pubmed |
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