pubmed-article:6770026 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C1536403 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0022686 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0003342 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0443286 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0041361 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0086344 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C0470187 | lld:lifeskim |
pubmed-article:6770026 | lifeskim:mentions | umls-concept:C1998811 | lld:lifeskim |
pubmed-article:6770026 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:6770026 | pubmed:dateCreated | 1980-8-15 | lld:pubmed |
pubmed-article:6770026 | pubmed:abstractText | Cytolytic T lymphocytes (CTL) specific for the virus-induced and leukemia-associated Friend, Moloney, Rauscher (FMR) antigen are easily detected in the spleens of primary and secondary stimulated H-2b or H-2d mice. They react, respectively, with H-2Db + FMR and H-2Kd + FMR; Dd and Kb never being involved. On the other hand, recombinant (KbDd) mice are relatively low responders that produce CTL only after secondary stimulation. Competition and blocking experiments with monospecific anti-H-2 antibodies have demonstrated that on the same H-2b tumor cells, C57BL/6 (H-2b) lymphocytes recognize Db + FMR, whereas B10.A(5R) lymphocytes recognize Kb + FMR, the restriction cannot, therefore be explained by a specific association of viral molecules with certain H-2 products. The CTL response of (B10 X 5R)F1 hybrids is (a) easily detected in primary reaction, the high responder anti-FMR phenotype being dominant and (b) directed against Db + FMR, F1 mice being low responder against Kb + FMR like the B10 parent. These results suggest that a D region-associated immune response gene controls the cell-mediated anti-FMR reaction, the best available H-2 + FMR antigenic association being chosen by CTL precursors. | lld:pubmed |
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pubmed-article:6770026 | pubmed:language | eng | lld:pubmed |
pubmed-article:6770026 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6770026 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:6770026 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6770026 | pubmed:month | Jun | lld:pubmed |
pubmed-article:6770026 | pubmed:issn | 0022-1007 | lld:pubmed |
pubmed-article:6770026 | pubmed:author | pubmed-author:GomardEE | lld:pubmed |
pubmed-article:6770026 | pubmed:author | pubmed-author:LévyJ PJP | lld:pubmed |
pubmed-article:6770026 | pubmed:author | pubmed-author:ColombaniM... | lld:pubmed |
pubmed-article:6770026 | pubmed:author | pubmed-author:HéninYY | lld:pubmed |
pubmed-article:6770026 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6770026 | pubmed:day | 1 | lld:pubmed |
pubmed-article:6770026 | pubmed:volume | 151 | lld:pubmed |
pubmed-article:6770026 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6770026 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6770026 | pubmed:pagination | 1468-76 | lld:pubmed |
pubmed-article:6770026 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:6770026 | pubmed:meshHeading | pubmed-meshheading:6770026-... | lld:pubmed |
pubmed-article:6770026 | pubmed:year | 1980 | lld:pubmed |
pubmed-article:6770026 | pubmed:articleTitle | Immune response genes control T killer cell response against Moloney tumor antigen cytolysis regulating reactions against the best available H-2 + viral antigen association. | lld:pubmed |
pubmed-article:6770026 | pubmed:publicationType | Journal Article | lld:pubmed |
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