| pubmed-article:6456062 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6456062 | lifeskim:mentions | umls-concept:C1257890 | lld:lifeskim |
| pubmed-article:6456062 | lifeskim:mentions | umls-concept:C0037993 | lld:lifeskim |
| pubmed-article:6456062 | lifeskim:mentions | umls-concept:C0229664 | lld:lifeskim |
| pubmed-article:6456062 | lifeskim:mentions | umls-concept:C0019829 | lld:lifeskim |
| pubmed-article:6456062 | lifeskim:mentions | umls-concept:C1518071 | lld:lifeskim |
| pubmed-article:6456062 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:6456062 | pubmed:dateCreated | 1981-11-18 | lld:pubmed |
| pubmed-article:6456062 | pubmed:abstractText | A panel of monoclonal antibodies with well-defined specificities were used as probes to investigate the phenotypes and lineage of circulating lymphoid cells and splenocytes in untreated patients with Hodgkin's disease. A significant relative and absolute reduction in T cells (anti-T3+) was found only in patients with B-symptoms. There was no alteration in the fraction of helper (anti-T4+) or cytotoxic/suppressor (anti-T5+) T cells circulating in peripheral blood when compared to normals, nor was there activation of these cells as measured by the development of surface Ia (anti-I1+). Circulating T cell subsets were not altered 5 to 14 days after splenectomy. Splenic T cells were increased equally in involved and uninvolved spleen when compared with control spleens obtained from accident victims. These findings indicate that abnormal T cell function in Hodgkin's disease may be the result of subtle alterations in T cells or non-T immunoregulatory mechanisms. | lld:pubmed |
| pubmed-article:6456062 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6456062 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:6456062 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6456062 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6456062 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:6456062 | pubmed:issn | 0008-543X | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:SchlossmanS... | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:ReinherzE LEL | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:RosenthalD... | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:NadlerL MLM | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:BreardJJ | lld:pubmed |
| pubmed-article:6456062 | pubmed:author | pubmed-author:PosnerM RMR | lld:pubmed |
| pubmed-article:6456062 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6456062 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:6456062 | pubmed:volume | 48 | lld:pubmed |
| pubmed-article:6456062 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6456062 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6456062 | pubmed:pagination | 1170-6 | lld:pubmed |
| pubmed-article:6456062 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
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| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:meshHeading | pubmed-meshheading:6456062-... | lld:pubmed |
| pubmed-article:6456062 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:6456062 | pubmed:articleTitle | Lymphoid subpopulations of peripheral blood and spleen in untreated Hodgkin's disease. | lld:pubmed |
| pubmed-article:6456062 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6456062 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:6456062 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:6456062 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:6456062 | lld:pubmed |
