pubmed-article:6410799 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6410799 | lifeskim:mentions | umls-concept:C0001811 | lld:lifeskim |
pubmed-article:6410799 | lifeskim:mentions | umls-concept:C0002395 | lld:lifeskim |
pubmed-article:6410799 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
pubmed-article:6410799 | lifeskim:mentions | umls-concept:C0032744 | lld:lifeskim |
pubmed-article:6410799 | lifeskim:mentions | umls-concept:C0233929 | lld:lifeskim |
pubmed-article:6410799 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:6410799 | pubmed:dateCreated | 1983-9-20 | lld:pubmed |
pubmed-article:6410799 | pubmed:abstractText | In this study the positron emission tomographic (PET)-18F-2-deoxy-2-fluoro-D-glucose (FDG) technique was used to study both normal aging and senile dementia. The results derived from 15 young normal subjects (mean age, 26 +/- 5 years) and 22 elderly normal subjects (mean age, 66 +/- 7 years) failed to indicate significant metabolic changes associated with age. A group of 24 patients with senile dementia (mean age, 73 +/- 7 years) showed consistent diminutions in regional glucose use relative to the elderly normals. Across all brain regions the diminutions were 17%-24%. There were also significant correlations between the measures of glucose use and the measures of cognitive functioning. Discriminant function classification analysis results indicate that better than 80% classification accuracy can be achieved for individual PET measures. These data suggest a possible future diagnostic use of PET in senile dementia. | lld:pubmed |
pubmed-article:6410799 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6410799 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6410799 | pubmed:language | eng | lld:pubmed |
pubmed-article:6410799 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6410799 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:6410799 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6410799 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6410799 | pubmed:issn | 0195-6108 | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:YonekuraYY | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:EmmerichMM | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:FowlerJ SJS | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:KricheffI III | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:ReisbergBB | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:GeorgeA EAE | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:FerrisS HSH | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:de LeonM JMJ | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:WolfA PAP | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:BrodieJJ | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:WUK PKP | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:ChristmanD... | lld:pubmed |
pubmed-article:6410799 | pubmed:author | pubmed-author:GentesCC | lld:pubmed |
pubmed-article:6410799 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6410799 | pubmed:volume | 4 | lld:pubmed |
pubmed-article:6410799 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6410799 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6410799 | pubmed:pagination | 568-71 | lld:pubmed |
pubmed-article:6410799 | pubmed:dateRevised | 2008-2-14 | lld:pubmed |
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pubmed-article:6410799 | pubmed:articleTitle | Positron emission tomographic studies of aging and Alzheimer disease. | lld:pubmed |
pubmed-article:6410799 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:6410799 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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