| pubmed-article:6332633 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C0021755 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C0598894 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C1441547 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C1326347 | lld:lifeskim |
| pubmed-article:6332633 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:6332633 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:6332633 | pubmed:dateCreated | 1984-10-24 | lld:pubmed |
| pubmed-article:6332633 | pubmed:abstractText | Human interleukin-1 (IL-1), free of contaminating lymphokines, was isolated from cultures of purified monoblasts from a patient with acute monocytic leukemia. Partially purified IL-1 (diafiltration, ultrafiltration, and isoelectric focusing) stimulated proliferation of subconfluent human fibroblasts in vitro. Further purification of IL-1 by high-resolution gel filtration- and anion exchange-high performance liquid chromatography revealed that fibroblast proliferation activity could not be separated from IL-1 activity (thymocyte proliferation), suggesting that both activities are the properties of a single molecule. Fibroblasts and thymocytes exhibited a similar sensitivity to the proliferative effects of IL-1. These findings suggest that macrophages participating in inflammatory reactions in vivo might release IL-1, which could function to expand fibroblast populations at sites of inflammatory reactions, by acting as a fibroblast growth factor. | lld:pubmed |
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| pubmed-article:6332633 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6332633 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6332633 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:6332633 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6332633 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:6332633 | pubmed:issn | 0004-3591 | lld:pubmed |
| pubmed-article:6332633 | pubmed:author | pubmed-author:KangA HAH | lld:pubmed |
| pubmed-article:6332633 | pubmed:author | pubmed-author:Postlethwaite... | lld:pubmed |
| pubmed-article:6332633 | pubmed:author | pubmed-author:LachmanL BLB | lld:pubmed |
| pubmed-article:6332633 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6332633 | pubmed:volume | 27 | lld:pubmed |
| pubmed-article:6332633 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6332633 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6332633 | pubmed:pagination | 995-1001 | lld:pubmed |
| pubmed-article:6332633 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:6332633 | pubmed:year | 1984 | lld:pubmed |
| pubmed-article:6332633 | pubmed:articleTitle | Induction of fibroblast proliferation by interleukin-1 derived from human monocytic leukemia cells. | lld:pubmed |
| pubmed-article:6332633 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6332633 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:6332633 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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