pubmed-article:6245271 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6245271 | lifeskim:mentions | umls-concept:C0038975 | lld:lifeskim |
pubmed-article:6245271 | lifeskim:mentions | umls-concept:C0018270 | lld:lifeskim |
pubmed-article:6245271 | lifeskim:mentions | umls-concept:C0439680 | lld:lifeskim |
pubmed-article:6245271 | lifeskim:mentions | umls-concept:C0596988 | lld:lifeskim |
pubmed-article:6245271 | lifeskim:mentions | umls-concept:C0439828 | lld:lifeskim |
pubmed-article:6245271 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:6245271 | pubmed:dateCreated | 1980-6-25 | lld:pubmed |
pubmed-article:6245271 | pubmed:abstractText | When viral growth in TC-7 cells is compared with that in the simian virus 40 (SV40) transformed CV-1 line C6 some mutants of SV40 deleted between 0.54 and 0.59 on the standard map (dl 54/59 mutants) give relative bursts similar to those of wild-type strain 776, whereas others grow markedly poorer in the untransformed cell. In general, viruses which are defective by this criterion have been found to produce neither a fragmentary small-t protein nor a mature small-t mRNA. | lld:pubmed |
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pubmed-article:6245271 | pubmed:language | eng | lld:pubmed |
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pubmed-article:6245271 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:6245271 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6245271 | pubmed:month | Mar | lld:pubmed |
pubmed-article:6245271 | pubmed:issn | 0022-538X | lld:pubmed |
pubmed-article:6245271 | pubmed:author | pubmed-author:ToppW CWC | lld:pubmed |
pubmed-article:6245271 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6245271 | pubmed:volume | 33 | lld:pubmed |
pubmed-article:6245271 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6245271 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6245271 | pubmed:pagination | 1208-10 | lld:pubmed |
pubmed-article:6245271 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:6245271 | pubmed:year | 1980 | lld:pubmed |
pubmed-article:6245271 | pubmed:articleTitle | Variable defectiveness for lytic growth of the dl 54/59 mutants of simian virus 40. | lld:pubmed |
pubmed-article:6245271 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:6245271 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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