| pubmed-article:6189952 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0085862 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0003261 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1299583 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0017263 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C0086344 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1546857 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1608386 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1556066 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1549571 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1619636 | lld:lifeskim |
| pubmed-article:6189952 | lifeskim:mentions | umls-concept:C1514873 | lld:lifeskim |
| pubmed-article:6189952 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:6189952 | pubmed:dateCreated | 1983-7-29 | lld:pubmed |
| pubmed-article:6189952 | pubmed:abstractText | The primary in vitro antibody response to TNP-Ficoll was found to be under H-2-restricted Ir gene control. Strains B10(H-2b), B10.A(H-2a), and B10.S(9R) (H-2t4) were consistently low responders while strains D2.GD(H-2g2), B10.GD(H-2g2), and B10.S(H-2s) were high responders. The in vitro TNP-Ficoll response in congenic recombinant and F1 hybrid mice demonstrated the requirement for complementation of two independent Ir genes. One Ir gene mapped in or to the left of the I-A subregion with high responder alleles being s or d. The second Ir gene mapped to the right of the I-E subregion and required b or s alleles for complementation. These results were further supported by the ability to block the TNP-Ficoll response by appropriate anti-Ia serum pretreatment of the antigen-presenting macrophages. When a structurally different polysaccharide antigen TNP-dextran was used, an identical pattern of restriction was observed. | lld:pubmed |
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| pubmed-article:6189952 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6189952 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6189952 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6189952 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6189952 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:6189952 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:6189952 | pubmed:author | pubmed-author:NiederhuberJ... | lld:pubmed |
| pubmed-article:6189952 | pubmed:author | pubmed-author:HillstromL... | lld:pubmed |
| pubmed-article:6189952 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6189952 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:6189952 | pubmed:volume | 157 | lld:pubmed |
| pubmed-article:6189952 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6189952 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6189952 | pubmed:pagination | 2002-16 | lld:pubmed |
| pubmed-article:6189952 | pubmed:dateRevised | 2010-9-10 | lld:pubmed |
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| pubmed-article:6189952 | pubmed:meshHeading | pubmed-meshheading:6189952-... | lld:pubmed |
| pubmed-article:6189952 | pubmed:year | 1983 | lld:pubmed |
| pubmed-article:6189952 | pubmed:articleTitle | IR gene regulation of the response to trinitrophenyl-polysaccharides. Two independent genes are required for antibody production. | lld:pubmed |
| pubmed-article:6189952 | pubmed:publicationType | Journal Article | lld:pubmed |
