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| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C0006675 | lld:lifeskim |
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| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C1179435 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C0017547 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C0037473 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C0001272 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C1705248 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C1548799 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C1524073 | lld:lifeskim |
| pubmed-article:5723516 | lifeskim:mentions | umls-concept:C0449432 | lld:lifeskim |
| pubmed-article:5723516 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:5723516 | pubmed:dateCreated | 1969-1-25 | lld:pubmed |
| pubmed-article:5723516 | pubmed:abstractText | 1. Action potentials resulting from direct stimulation can be recorded from the soma of the Aplysia giant neurone (located in the visceral ganglion) in sodium-free and in calcium-free external solutions. The neurones were impaled by internal micro-electrodes throughout the change of external solutions.2. Complete replacement of either sodium or calcium in the bathing medium with Tris results in only a partial reduction of spike overshoot. Simultaneous replacement of both sodium and calcium reversibly and quickly abolishes the spike.3. The sodium component of the spike in a calcium-free medium is blocked by tetrodotoxin; the drug has no effect on the calcium-dependent spike in sodium-free medium. Externally applied cobalt chloride blocks only the calcium-dependent component.4. In calcium-free media, the overshoot value varies with sodium concentration in the manner predicted for a sodium electrode. In sodium-free media, the membrane behaves like a calcium electrode.5. These results suggest that, during the normal action potential, both sodium and calcium act as carriers of the inward-directed current. | lld:pubmed |
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| pubmed-article:5723516 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:5723516 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:5723516 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:5723516 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:5723516 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:5723516 | pubmed:author | pubmed-author:JungeDD | lld:pubmed |
| pubmed-article:5723516 | pubmed:author | pubmed-author:GeduldigDD | lld:pubmed |
| pubmed-article:5723516 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:5723516 | pubmed:volume | 199 | lld:pubmed |
| pubmed-article:5723516 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:5723516 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:5723516 | pubmed:pagination | 347-65 | lld:pubmed |
| pubmed-article:5723516 | pubmed:dateRevised | 2010-9-10 | lld:pubmed |
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| pubmed-article:5723516 | pubmed:year | 1968 | lld:pubmed |
| pubmed-article:5723516 | pubmed:articleTitle | Sodium and calcium components of action potentials in the Aplysia giant neurone. | lld:pubmed |
| pubmed-article:5723516 | pubmed:publicationType | Journal Article | lld:pubmed |
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