| pubmed-article:5435494 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:5435494 | lifeskim:mentions | umls-concept:C0221102 | lld:lifeskim |
| pubmed-article:5435494 | lifeskim:mentions | umls-concept:C0016581 | lld:lifeskim |
| pubmed-article:5435494 | lifeskim:mentions | umls-concept:C0042847 | lld:lifeskim |
| pubmed-article:5435494 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:5435494 | lifeskim:mentions | umls-concept:C0392760 | lld:lifeskim |
| pubmed-article:5435494 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:5435494 | pubmed:dateCreated | 1970-5-4 | lld:pubmed |
| pubmed-article:5435494 | pubmed:abstractText | 1. Formiminoglutamic acid, a product of the catabolism of histidine, is excreted in abnormally large amounts in the urines of vitamin B(12)-deficient rats and of vitamin B(12)-deficient sheep; the excretion is reduced to negligible amounts after administration of vitamin B(12). 2. After administration of certain methyl donors to vitamin B(12)-deficient rats or sheep urinary excretion of formiminoglutamic acid is temporarily decreased. 3. Irrespective of the pteroylglutamic acid status of the animals neither vitamin B(12)-deficient rats nor vitamin B(12)-deficient sheep have the ability to deal efficiently with histidine. 4. In sheep, urinary excretion of formiminoglutamic acid is increased after administration of aminopterin; treatment with pteroylglutamic acid restores the ability of the animal to deal with the catabolic products of histidine. 5. The possible functions of vitamin B(12) and methionine in relieving a virtual deficiency of pteroylglutamic acid are discussed. | lld:pubmed |
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| pubmed-article:5435494 | pubmed:language | eng | lld:pubmed |
| pubmed-article:5435494 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:5435494 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:5435494 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:5435494 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:5435494 | pubmed:issn | 0264-6021 | lld:pubmed |
| pubmed-article:5435494 | pubmed:author | pubmed-author:AllenS HSH | lld:pubmed |
| pubmed-article:5435494 | pubmed:author | pubmed-author:MarstonH RHR | lld:pubmed |
| pubmed-article:5435494 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:5435494 | pubmed:volume | 116 | lld:pubmed |
| pubmed-article:5435494 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:5435494 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:5435494 | pubmed:pagination | 681-8 | lld:pubmed |
| pubmed-article:5435494 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:5435494 | pubmed:year | 1970 | lld:pubmed |
| pubmed-article:5435494 | pubmed:articleTitle | Factors affecting formiminoglutamic acid excretion in vitamin B 12 deficiency. | lld:pubmed |
| pubmed-article:5435494 | pubmed:publicationType | Journal Article | lld:pubmed |
