| pubmed-article:5145907 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C0004651 | lld:lifeskim |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C1095909 | lld:lifeskim |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C0038774 | lld:lifeskim |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C0441472 | lld:lifeskim |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C1412438 | lld:lifeskim |
| pubmed-article:5145907 | lifeskim:mentions | umls-concept:C1412444 | lld:lifeskim |
| pubmed-article:5145907 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:5145907 | pubmed:dateCreated | 1972-6-21 | lld:pubmed |
| pubmed-article:5145907 | pubmed:abstractText | Bacteriophage mu2 is inactivated by both mono- and di-functional sulphur mustards at relatively low extents of alkylation. No degradation of alkylated RNA was detected. Cross-linking of RNA to protein was observed with the difunctional agent, but this reaction was only a minor contribution to the inactivation. Analyses of the reaction products in bacteriophage RNA showed that, at the mean lethal doses, more than one mono-alkylation of guanine had occurred but the sum total of other types of RNA alkylation was close to a single event. The results therefore suggest that inactivation results from the mono-alkylation of adenine or cytosine. In experiments with the difunctional agent cross-linking of RNA bases or of RNA to protein also prevented replication, the existence of these reactions accounting for the greater sensitivity of the bacteriophage to this agent. | lld:pubmed |
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| pubmed-article:5145907 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:5145907 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:5145907 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:5145907 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:5145907 | pubmed:issn | 0264-6021 | lld:pubmed |
| pubmed-article:5145907 | pubmed:author | pubmed-author:EdwardsP APA | lld:pubmed |
| pubmed-article:5145907 | pubmed:author | pubmed-author:LawleyP DPD | lld:pubmed |
| pubmed-article:5145907 | pubmed:author | pubmed-author:ShooterK VKV | lld:pubmed |
| pubmed-article:5145907 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:5145907 | pubmed:volume | 125 | lld:pubmed |
| pubmed-article:5145907 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:5145907 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:5145907 | pubmed:pagination | 829-40 | lld:pubmed |
| pubmed-article:5145907 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:5145907 | pubmed:year | 1971 | lld:pubmed |
| pubmed-article:5145907 | pubmed:articleTitle | The action of mono- and di-functional sulphur mustards on the ribonucleic acid-containing bacteriophage mu2. | lld:pubmed |
| pubmed-article:5145907 | pubmed:publicationType | Journal Article | lld:pubmed |
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