| pubmed-article:457258 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0025011 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0021311 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0018873 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0042542 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0205322 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C2346501 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C0439828 | lld:lifeskim |
| pubmed-article:457258 | lifeskim:mentions | umls-concept:C1334043 | lld:lifeskim |
| pubmed-article:457258 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:457258 | pubmed:dateCreated | 1979-9-17 | lld:pubmed |
| pubmed-article:457258 | pubmed:abstractText | The HeLa subline K11A-HG-1 (line of HeLa cells persistently infected with Edomonston measles virus but containing little or no transmissible infectious virus) was co-cultivated with Vero cells. Focal syncytia were formed containing measles antigen and accumulations of nucleocapsid-like structures with no detectable production of transmissible infectious virus or positive hemadsorption. The infection aborted between 2 and 3 weeks after preparation of co-cultures. Upon subculture of co-cultures, occasionally complete infections (progressive syncytial degeneration, hemadsorption, and production of transmissible infectious virus) appeared. A linear dose response curve for nontransmissible infection was obtained along with evidence that measles antigen had to be present on the surface of K11A-HG-1 cells for their infectivity for Vero cells. The basis for initiation of Vero cell infection by living K11A-HG-1 cells, but not by nonviable intact K11A-HG-1 cells killed by a virus-preserving technique, nor by disrupted K11A-HG-1 cells, is, at present, a matter of speculation. However, several lines of evidence were obtained which suggested that subsequent development of delayed variable transmissible Vero cell infection occurred because of a type of viral interference, including the presence of an inhibitor in K11A-HG-1 cultures, the bulk of which was cell-associated. | lld:pubmed |
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| pubmed-article:457258 | pubmed:language | eng | lld:pubmed |
| pubmed-article:457258 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:457258 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:457258 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:457258 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:457258 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:457258 | pubmed:author | pubmed-author:RustigianRR | lld:pubmed |
| pubmed-article:457258 | pubmed:author | pubmed-author:DarlingtonR... | lld:pubmed |
| pubmed-article:457258 | pubmed:author | pubmed-author:WinstonS HSH | lld:pubmed |
| pubmed-article:457258 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:457258 | pubmed:volume | 23 | lld:pubmed |
| pubmed-article:457258 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:457258 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:457258 | pubmed:pagination | 775-86 | lld:pubmed |
| pubmed-article:457258 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:457258 | pubmed:meshHeading | pubmed-meshheading:457258-S... | lld:pubmed |
| pubmed-article:457258 | pubmed:year | 1979 | lld:pubmed |
| pubmed-article:457258 | pubmed:articleTitle | Variable infection of Vero cells and homologous interference after co-cultivation with HeLa cells with persistent defective infection by Edmonston measles virus. | lld:pubmed |
| pubmed-article:457258 | pubmed:publicationType | Journal Article | lld:pubmed |
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