pubmed-article:4556464 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0020663 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0008810 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0013124 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0026606 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
pubmed-article:4556464 | lifeskim:mentions | umls-concept:C0849355 | lld:lifeskim |
pubmed-article:4556464 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:4556464 | pubmed:dateCreated | 1972-8-19 | lld:pubmed |
pubmed-article:4556464 | pubmed:abstractText | Bilateral electrolytic lesions in the suprachiasmatic nuclei permanently eliminated nocturnal and circadian rhythms in drinking behavior and locomotor activity of albino rats. The generation of 24-hr behavioral rhythms and the entrainment of these rhythms to the light-dark cycle of environmental illumination may be coordinated by neurons in the suprachiasmatic region of the rat brain. Destruction of the medial preoptic area had no effect on 24-hr drinking rhythms. | lld:pubmed |
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pubmed-article:4556464 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:4556464 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:4556464 | pubmed:language | eng | lld:pubmed |
pubmed-article:4556464 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:4556464 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:4556464 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:4556464 | pubmed:month | Jun | lld:pubmed |
pubmed-article:4556464 | pubmed:issn | 0027-8424 | lld:pubmed |
pubmed-article:4556464 | pubmed:author | pubmed-author:StephanF KFK | lld:pubmed |
pubmed-article:4556464 | pubmed:author | pubmed-author:ZuckerII | lld:pubmed |
pubmed-article:4556464 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:4556464 | pubmed:volume | 69 | lld:pubmed |
pubmed-article:4556464 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:4556464 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:4556464 | pubmed:pagination | 1583-6 | lld:pubmed |
pubmed-article:4556464 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
pubmed-article:4556464 | pubmed:meshHeading | pubmed-meshheading:4556464-... | lld:pubmed |
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pubmed-article:4556464 | pubmed:meshHeading | pubmed-meshheading:4556464-... | lld:pubmed |
pubmed-article:4556464 | pubmed:year | 1972 | lld:pubmed |
pubmed-article:4556464 | pubmed:articleTitle | Circadian rhythms in drinking behavior and locomotor activity of rats are eliminated by hypothalamic lesions. | lld:pubmed |
pubmed-article:4556464 | pubmed:publicationType | Journal Article | lld:pubmed |
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