pubmed-article:4139235 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:4139235 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:4139235 | lifeskim:mentions | umls-concept:C0061472 | lld:lifeskim |
pubmed-article:4139235 | lifeskim:mentions | umls-concept:C0003261 | lld:lifeskim |
pubmed-article:4139235 | lifeskim:mentions | umls-concept:C0017263 | lld:lifeskim |
pubmed-article:4139235 | lifeskim:mentions | umls-concept:C0220839 | lld:lifeskim |
pubmed-article:4139235 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:4139235 | pubmed:dateCreated | 1975-2-18 | lld:pubmed |
pubmed-article:4139235 | pubmed:abstractText | In order to further delineate the mechanisms underlying genetic unresponsiveness, tetraparental mice were constructed from immune response-1A gene high responder and low responder parental genotypes, then were immunized with poly-L-(Tyr,Glu)-poly-D,L-Ala--poly-L-Lys ((T,G)-A--L). An analysis of the total serum allotype mixture and of the antigen-binding capacity of the separated allotypes demonstrated that in the milieu of a tetraparental mouse, both high and low responder B cells could be stimulated equally to produce identical high titered anti-(T,G)-A--L responses. Furthermore, these studies show that effective stimulation could occur across a histocompatibility disparity. | lld:pubmed |
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pubmed-article:4139235 | pubmed:language | eng | lld:pubmed |
pubmed-article:4139235 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:4139235 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:4139235 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:4139235 | pubmed:month | Dec | lld:pubmed |
pubmed-article:4139235 | pubmed:issn | 0022-1007 | lld:pubmed |
pubmed-article:4139235 | pubmed:author | pubmed-author:HerzenbergL... | lld:pubmed |
pubmed-article:4139235 | pubmed:author | pubmed-author:FreedJ HJH | lld:pubmed |
pubmed-article:4139235 | pubmed:author | pubmed-author:McDevittH OHO | lld:pubmed |
pubmed-article:4139235 | pubmed:author | pubmed-author:BechtolK BKB | lld:pubmed |
pubmed-article:4139235 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:4139235 | pubmed:day | 1 | lld:pubmed |
pubmed-article:4139235 | pubmed:volume | 140 | lld:pubmed |
pubmed-article:4139235 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:4139235 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:4139235 | pubmed:pagination | 1660-75 | lld:pubmed |
pubmed-article:4139235 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:4139235 | pubmed:year | 1974 | lld:pubmed |
pubmed-article:4139235 | pubmed:articleTitle | Genetic control of the antibody response to poly-L(Tyr,Glu)-poly-D,L-Ala--poly-L-Lys in C3H--CWB tetraparental mice. | lld:pubmed |
pubmed-article:4139235 | pubmed:publicationType | Journal Article | lld:pubmed |
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