pubmed-article:3987916 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3987916 | lifeskim:mentions | umls-concept:C1705480 | lld:lifeskim |
pubmed-article:3987916 | lifeskim:mentions | umls-concept:C0206162 | lld:lifeskim |
pubmed-article:3987916 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
pubmed-article:3987916 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:3987916 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:3987916 | pubmed:dateCreated | 1985-6-10 | lld:pubmed |
pubmed-article:3987916 | pubmed:abstractText | Our data indicate that vasopressin facilitates baroreflex inhibition of lumbar sympathetic nerve activity by two mechanisms: it sensitizes baroreceptors locally and shifts the stimulus-response curve so that a lower carotid sinus pressure results in a certain level of reflex sympathetic inhibition; it also produces a corresponding shift when given i.v. and excluded from baroreceptors implicating a second, central mechanism for facilitation of baroreflexes. In contrast, angiotensin II attenuates baroreflex inhibition of peripheral sympathetic function and this is accounted for totally by a central action. Why these differences occur present challenging new questions for future investigation. | lld:pubmed |
pubmed-article:3987916 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:language | eng | lld:pubmed |
pubmed-article:3987916 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:3987916 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3987916 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3987916 | pubmed:month | May | lld:pubmed |
pubmed-article:3987916 | pubmed:issn | 0014-9446 | lld:pubmed |
pubmed-article:3987916 | pubmed:author | pubmed-author:AbboudF MFM | lld:pubmed |
pubmed-article:3987916 | pubmed:author | pubmed-author:SchmidP GPG | lld:pubmed |
pubmed-article:3987916 | pubmed:author | pubmed-author:OtaL HLH | lld:pubmed |
pubmed-article:3987916 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3987916 | pubmed:volume | 44 | lld:pubmed |
pubmed-article:3987916 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3987916 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3987916 | pubmed:pagination | 2388-92 | lld:pubmed |
pubmed-article:3987916 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
pubmed-article:3987916 | pubmed:meshHeading | pubmed-meshheading:3987916-... | lld:pubmed |
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pubmed-article:3987916 | pubmed:meshHeading | pubmed-meshheading:3987916-... | lld:pubmed |
pubmed-article:3987916 | pubmed:meshHeading | pubmed-meshheading:3987916-... | lld:pubmed |
pubmed-article:3987916 | pubmed:year | 1985 | lld:pubmed |
pubmed-article:3987916 | pubmed:articleTitle | Different effects of vasopressin and angiotensin II on baroreflexes. | lld:pubmed |
pubmed-article:3987916 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:3987916 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:3987916 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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