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| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0596995 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0001271 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C1457869 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C1705535 | lld:lifeskim |
| pubmed-article:3785229 | lifeskim:mentions | umls-concept:C2728259 | lld:lifeskim |
| pubmed-article:3785229 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:3785229 | pubmed:dateCreated | 1986-12-22 | lld:pubmed |
| pubmed-article:3785229 | pubmed:abstractText | The rat myogenic cell line L6E9 induces skeletal but not cardiac alpha-actin mRNA upon fusion to form myotubes. However, when a human cardiac alpha-actin gene was introduced into L6E9 myoblasts, differentiation of the cells led to the accumulation of human gene transcripts in parallel with those derived from the endogenous skeletal alpha-actin gene. This result demonstrates that factors which direct rat myogenesis can regulate a muscle gene from another species and that the L6E9 cells may have a defect in their ability to activate endogenous cardiac actin gene expression. | lld:pubmed |
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| pubmed-article:3785229 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:3785229 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3785229 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3785229 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:3785229 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:3785229 | pubmed:author | pubmed-author:SkoultchiAA | lld:pubmed |
| pubmed-article:3785229 | pubmed:author | pubmed-author:KedesLL | lld:pubmed |
| pubmed-article:3785229 | pubmed:author | pubmed-author:HickeyRR | lld:pubmed |
| pubmed-article:3785229 | pubmed:author | pubmed-author:GunningPP | lld:pubmed |
| pubmed-article:3785229 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3785229 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:3785229 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3785229 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3785229 | pubmed:pagination | 3287-90 | lld:pubmed |
| pubmed-article:3785229 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3785229 | pubmed:meshHeading | pubmed-meshheading:3785229-... | lld:pubmed |
| pubmed-article:3785229 | pubmed:year | 1986 | lld:pubmed |
| pubmed-article:3785229 | pubmed:articleTitle | Regulation of a human cardiac actin gene introduced into rat L6 myoblasts suggests a defect in their myogenic program. | lld:pubmed |
| pubmed-article:3785229 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3785229 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3785229 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:3785229 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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