| pubmed-article:3658812 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C0030520 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C0012359 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C0486805 | lld:lifeskim |
| pubmed-article:3658812 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:3658812 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:3658812 | pubmed:dateCreated | 1987-10-30 | lld:pubmed |
| pubmed-article:3658812 | pubmed:abstractText | The in vivo responsiveness of small arterioles to the topical administration of two parathyroid hormone fragments was investigated using television microscopy. Male Sprague-Dawley rats were anesthetized with sodium pentobarbital (50 mg/kg) and second- and third-order arterioles in the cremaster muscle were exposed to increasing concentrations (2 X 10(-5) to 6 X 10(-4) mg/ml) of either hPTH (1-34) or bPTH-(3-34). Second- and third-order arterioles within the cremaster dilated (183% and 281% of control, respectively) following exposure to PTH-(1-34) in bath concentration of 10(-4) mg/ml and above. The dilation associated with PTH administration was abolished in second-order and greatly attenuated for third-order arterioles when the first two amino acid residues of the PTH molecule were removed (PTH (3-34) fragment). Inhibition of endogenous prostaglandins synthesis with mefenamic acid did not attenuate the vasodilator response to PTH. However, exposure to the muscarinic blocking agent atropine (10(-7) g/ml) totally inhibited the dilator response to PTH-(1-34). These data suggest that PTH induces arteriolar dilation by stimulation of muscarinic receptors in the vasculature possibly by causing the release of endogenous acetylcholine. | lld:pubmed |
| pubmed-article:3658812 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3658812 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3658812 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3658812 | pubmed:issn | 0196-9781 | lld:pubmed |
| pubmed-article:3658812 | pubmed:author | pubmed-author:JoshuaI GIG | lld:pubmed |
| pubmed-article:3658812 | pubmed:author | pubmed-author:FossJJ | lld:pubmed |
| pubmed-article:3658812 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3658812 | pubmed:volume | 8 | lld:pubmed |
| pubmed-article:3658812 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3658812 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3658812 | pubmed:pagination | 443-8 | lld:pubmed |
| pubmed-article:3658812 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
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| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:meshHeading | pubmed-meshheading:3658812-... | lld:pubmed |
| pubmed-article:3658812 | pubmed:articleTitle | In vivo arteriolar dilation in response to parathyroid hormone fragments. | lld:pubmed |
| pubmed-article:3658812 | pubmed:affiliation | Department of Physiology and Biophysics, University of Louisville, KY 40292. | lld:pubmed |
| pubmed-article:3658812 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3658812 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:3658812 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
