| pubmed-article:3607365 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0034493 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0027882 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0295352 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0015127 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0549255 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C1314792 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0449560 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:3607365 | lifeskim:mentions | umls-concept:C0439831 | lld:lifeskim |
| pubmed-article:3607365 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:3607365 | pubmed:dateCreated | 1987-9-16 | lld:pubmed |
| pubmed-article:3607365 | pubmed:abstractText | Intracellular recordings were made from parasympathetic neurones of the rabbit vesical pelvic ganglia maintained in vitro. 5-Hydroxytryptamine (5-HT) caused a membrane depolarization which was antagonized by ICS 205-930 ([3 alpha-tropanyl]-1H-indole-3-carboxylic acid ester) but not by methysergide. ICS 205-930 caused a parallel shift to the right of the dose-response curve for 5-HT. These results suggest that the 5-HT3 receptor is involved in the membrane depolarization. | lld:pubmed |
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| pubmed-article:3607365 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3607365 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3607365 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3607365 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3607365 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:3607365 | pubmed:issn | 0007-1188 | lld:pubmed |
| pubmed-article:3607365 | pubmed:author | pubmed-author:AkasuTT | lld:pubmed |
| pubmed-article:3607365 | pubmed:author | pubmed-author:TokimasaTT | lld:pubmed |
| pubmed-article:3607365 | pubmed:author | pubmed-author:HasuoHH | lld:pubmed |
| pubmed-article:3607365 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3607365 | pubmed:volume | 91 | lld:pubmed |
| pubmed-article:3607365 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3607365 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3607365 | pubmed:pagination | 453-5 | lld:pubmed |
| pubmed-article:3607365 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:3607365 | pubmed:meshHeading | pubmed-meshheading:3607365-... | lld:pubmed |
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| pubmed-article:3607365 | pubmed:meshHeading | pubmed-meshheading:3607365-... | lld:pubmed |
| pubmed-article:3607365 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:3607365 | pubmed:articleTitle | Activation of 5-HT3 receptor subtypes causes rapid excitation of rabbit parasympathetic neurones. | lld:pubmed |
| pubmed-article:3607365 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3607365 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:3607365 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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