pubmed-article:3326441 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C0006840 | lld:lifeskim |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C0002679 | lld:lifeskim |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C0205125 | lld:lifeskim |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C1708528 | lld:lifeskim |
pubmed-article:3326441 | lifeskim:mentions | umls-concept:C0599918 | lld:lifeskim |
pubmed-article:3326441 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:3326441 | pubmed:dateCreated | 1988-4-5 | lld:pubmed |
pubmed-article:3326441 | pubmed:abstractText | Previous observations suggest that tubulo-glomerular feedback could be involved in amphotericin B nephrotoxicity. We then investigated the influence of sodium status on the occurrence of renal damage during amphotericin B therapy. A retrospective survey demonstrated that impaired renal function occurred during therapy in 67 per cent of the patients who received amphotericin B alone and in 12 per cent of the patients who received amphotericin B and ticarcillin (parenteral sodium supplement of 100-150 meq per day). Prospective studies were then undertaken both in adults and children. Intravenous sodium supplement was given intravenously as routine prophylaxis with amphotericin B therapy. In all courses amphotericin B was successfully administered without deterioration in renal function. These results support the hypothesis that parenteral sodium supplementation reduces the frequency of developing impaired renal function during amphotericin B therapy. | lld:pubmed |
pubmed-article:3326441 | pubmed:language | fre | lld:pubmed |
pubmed-article:3326441 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3326441 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:3326441 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3326441 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3326441 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3326441 | pubmed:issn | 0003-3898 | lld:pubmed |
pubmed-article:3326441 | pubmed:author | pubmed-author:BranchR ARA | lld:pubmed |
pubmed-article:3326441 | pubmed:author | pubmed-author:AujardYY | lld:pubmed |
pubmed-article:3326441 | pubmed:author | pubmed-author:HeidemannHH | lld:pubmed |
pubmed-article:3326441 | pubmed:author | pubmed-author:JacqzEE | lld:pubmed |
pubmed-article:3326441 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3326441 | pubmed:volume | 45 | lld:pubmed |
pubmed-article:3326441 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3326441 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3326441 | pubmed:pagination | 689-93 | lld:pubmed |
pubmed-article:3326441 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:3326441 | pubmed:year | 1987 | lld:pubmed |
pubmed-article:3326441 | pubmed:articleTitle | [Prevention of nephrotoxicity of amphotericin B during the treatment of deep candidiasis]. | lld:pubmed |
pubmed-article:3326441 | pubmed:affiliation | Service de Néonatologie, Hôpital Bretonneau, Paris. | lld:pubmed |
pubmed-article:3326441 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:3326441 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:3326441 | pubmed:publicationType | English Abstract | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:3326441 | lld:pubmed |