| pubmed-article:322150 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C0229671 | lld:lifeskim |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C0024264 | lld:lifeskim |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:322150 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:322150 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:322150 | pubmed:dateCreated | 1977-5-12 | lld:pubmed |
| pubmed-article:322150 | pubmed:abstractText | Serum from bacillus Calmette-Guerin-infected mice injected with endotoxin induces the appearance of surface immunoglobulin, Ia antigen, and complement receptor on the surface of precursor bone-marrow-derived (B) cells. While endotoxin itself causes phenotypic conversion of both thymus-derived (T) cells and B cells in vitro, the endotoxin-induced serum factor was found to be a selective inducer of B cell differentiation. Spleen cells rendered immunodeficient by removal of B cells bearing the complement receptor regained the capacity to cooperate with helper T cells and to produce antibody against red cell antigens in vitro upon upon addition of the serum factor to the culture medium. Thus, a factor that controls selective phenotypic and functional differentiation of B cells has been identified and can now be characterized, | lld:pubmed |
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| pubmed-article:322150 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:322150 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:322150 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:322150 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:322150 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:322150 | pubmed:author | pubmed-author:HoffmannM KMK | lld:pubmed |
| pubmed-article:322150 | pubmed:author | pubmed-author:OldL JLJ | lld:pubmed |
| pubmed-article:322150 | pubmed:author | pubmed-author:OettgenH FHF | lld:pubmed |
| pubmed-article:322150 | pubmed:author | pubmed-author:HammerlingUU | lld:pubmed |
| pubmed-article:322150 | pubmed:author | pubmed-author:ChinA FAF | lld:pubmed |
| pubmed-article:322150 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:322150 | pubmed:volume | 74 | lld:pubmed |
| pubmed-article:322150 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:322150 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:322150 | pubmed:pagination | 1200-3 | lld:pubmed |
| pubmed-article:322150 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:322150 | pubmed:year | 1977 | lld:pubmed |
| pubmed-article:322150 | pubmed:articleTitle | Endotoxin-induced serum factor controlling differentiation of bone-marrow-derived lymphocytes. | lld:pubmed |
| pubmed-article:322150 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:322150 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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