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| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1373200 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1705175 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1882348 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1705178 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1705176 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1546465 | lld:lifeskim |
| pubmed-article:3197958 | lifeskim:mentions | umls-concept:C1705177 | lld:lifeskim |
| pubmed-article:3197958 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:3197958 | pubmed:dateCreated | 1989-1-13 | lld:pubmed |
| pubmed-article:3197958 | pubmed:abstractText | The t region of mouse chromosome 17 exhibits recombination suppression with wild-type chromatin. However, the region has resisted classical genetic dissection because of a lack of defined variants. Mutations induced by N-ethyl-N-nitrosourea (ENU) at the Brachyury (T), quaking (qk), and tufted (tf) loci of the mouse tw5 haplotype have now allowed the analysis of crossovers between two complete t haplotypes. A classical breeding analysis of the complete t haplotypes, tw5 and t12, utilizing the newly induced markers, reveals two inversions in t chromatin: one involving T and qk, and one involving tf and the H-2 complex. Moreover, the recombination frequency between the loci of T and qk is reduced compared to the frequency reported in normal chromatin. These two inversions are a sufficient explanation for the recombination inhibition with normal chromatin exhibited by t haplotypes isolated from the wild. Furthermore, the reduced recombination frequency between T and qk may indicate that the proximal gene rearrangement is not a simple inversion. | lld:pubmed |
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| pubmed-article:3197958 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:3197958 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3197958 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3197958 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:3197958 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:3197958 | pubmed:author | pubmed-author:BodeV CVC | lld:pubmed |
| pubmed-article:3197958 | pubmed:author | pubmed-author:JusticeM JMJ | lld:pubmed |
| pubmed-article:3197958 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3197958 | pubmed:volume | 120 | lld:pubmed |
| pubmed-article:3197958 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3197958 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3197958 | pubmed:pagination | 533-43 | lld:pubmed |
| pubmed-article:3197958 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:3197958 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:3197958 | pubmed:articleTitle | Genetic analysis of mouse t haplotypes using mutations induced by ethylnitrosourea mutagenesis: the order of T and qk is inverted in t mutants. | lld:pubmed |
| pubmed-article:3197958 | pubmed:affiliation | Division of Biology, Kansas State University, Manhattan 66506. | lld:pubmed |
| pubmed-article:3197958 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3197958 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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