pubmed-article:3190554 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C0025266 | lld:lifeskim |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C0023796 | lld:lifeskim |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C0025519 | lld:lifeskim |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C0052190 | lld:lifeskim |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
pubmed-article:3190554 | lifeskim:mentions | umls-concept:C1547548 | lld:lifeskim |
pubmed-article:3190554 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:3190554 | pubmed:dateCreated | 1988-12-20 | lld:pubmed |
pubmed-article:3190554 | pubmed:abstractText | The C apolipoproteins are normally transferred to high density lipoproteins (HDL) after lipolysis of very low density lipoprotein (VLDL) triglyceride. In previous studies, a loss of plasma C apolipoproteins was documented after heparin-induced lipolysis in hypertriglyceridemic subjects. The present studies were designed to determine if this decline in plasma C apolipoproteins was due to their clearance with VLDL remnants. Five Type IV hypertriglyceridemic and two normal subjects were injected with 125I-VLDL and 131I-low density lipoproteins (LDL) to document kinetically an excess of VLDL apolipoprotein (apo) B flux relative to LDL apo B flux in the Type IV subjects. A mean of 46% VLDL apo B was cleared from the circulation, without conversion to intermediate density lipoprotein (IDL) or LDL. Heparin was then infused (9000 IU over 4 hours) to generate an excess of VLDL remnants that were not converted to IDL or LDL. VLDL triglyceride, apo B, and apo C concentrations fell at a similar rate. VLDL apo B declined by 42% (p less than 0.01). However, no increases were observed in IDL or LDL apo B in the Type IV subjects. This resulted in a 14% (p less than 0.01) decline in plasma apo B concentrations, indicating a clearance of VLDL remnants. VLDL apo C-II and C-III concentrations fell by 42% (p less than 0.025) and 52% (p less than 0.01), respectively. During the first 2.5 hours of infusion, they were almost quantitatively recovered in HDL. Thereafter, the C apolipoproteins declined in HDL during which time VLDL apo C concentrations continued to decline.(ABSTRACT TRUNCATED AT 250 WORDS) | lld:pubmed |
pubmed-article:3190554 | pubmed:language | eng | lld:pubmed |
pubmed-article:3190554 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3190554 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3190554 | pubmed:issn | 0276-5047 | lld:pubmed |
pubmed-article:3190554 | pubmed:author | pubmed-author:BreckenridgeW... | lld:pubmed |
pubmed-article:3190554 | pubmed:author | pubmed-author:WolfeB MBM | lld:pubmed |
pubmed-article:3190554 | pubmed:author | pubmed-author:HuffM WMW | lld:pubmed |
pubmed-article:3190554 | pubmed:author | pubmed-author:StrongW LWL | lld:pubmed |
pubmed-article:3190554 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3190554 | pubmed:volume | 8 | lld:pubmed |
pubmed-article:3190554 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3190554 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3190554 | pubmed:pagination | 471-9 | lld:pubmed |
pubmed-article:3190554 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:meshHeading | pubmed-meshheading:3190554-... | lld:pubmed |
pubmed-article:3190554 | pubmed:articleTitle | Metabolism of apolipoproteins C-II, C-III, and B in hypertriglyceridemic men. Changes after heparin-induced lipolysis. | lld:pubmed |
pubmed-article:3190554 | pubmed:affiliation | Department of Medicine, University Hospital, University of Western Ontario, London, Canada. | lld:pubmed |
pubmed-article:3190554 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:3190554 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:3190554 | lld:pubmed |