| pubmed-article:3133245 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3133245 | lifeskim:mentions | umls-concept:C0040132 | lld:lifeskim |
| pubmed-article:3133245 | lifeskim:mentions | umls-concept:C0020281 | lld:lifeskim |
| pubmed-article:3133245 | lifeskim:mentions | umls-concept:C0596235 | lld:lifeskim |
| pubmed-article:3133245 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:3133245 | lifeskim:mentions | umls-concept:C0079411 | lld:lifeskim |
| pubmed-article:3133245 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:3133245 | pubmed:dateCreated | 1988-8-1 | lld:pubmed |
| pubmed-article:3133245 | pubmed:abstractText | A thyroid particulate fraction contains an NADPH-dependent H2O2-generating enzyme which requires Ca2+ for activity. A Chaps solubilized extract of the thyroid particulate fraction partially purified by DEAE chromatography did not show a dependence on Ca2+ for activity. Preincubation of the particulate fraction with Ca2+ yielded a preparation insensitive to Ca2+. The non-particulate fraction obtained after incubation of the particles in the presence of Ca2+ was able to inhibit, in the presence of EGTA, the Ca2+-desensitized particulate fraction and the enzyme isolated on DEAE. It is concluded that the reversible Ca2+ activation of the NADPH-dependent H2O2 generation was modulated in porcine thyroid tissue by (a) calcium-releasable inhibitor protein(s). | lld:pubmed |
| pubmed-article:3133245 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3133245 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3133245 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3133245 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3133245 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:3133245 | pubmed:issn | 0014-5793 | lld:pubmed |
| pubmed-article:3133245 | pubmed:author | pubmed-author:PommierJJ | lld:pubmed |
| pubmed-article:3133245 | pubmed:author | pubmed-author:VirionAA | lld:pubmed |
| pubmed-article:3133245 | pubmed:author | pubmed-author:DupuyCC | lld:pubmed |
| pubmed-article:3133245 | pubmed:author | pubmed-author:DèmeDD | lld:pubmed |
| pubmed-article:3133245 | pubmed:author | pubmed-author:KaniewskiJJ | lld:pubmed |
| pubmed-article:3133245 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3133245 | pubmed:day | 6 | lld:pubmed |
| pubmed-article:3133245 | pubmed:volume | 233 | lld:pubmed |
| pubmed-article:3133245 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3133245 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3133245 | pubmed:pagination | 74-8 | lld:pubmed |
| pubmed-article:3133245 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:3133245 | pubmed:meshHeading | pubmed-meshheading:3133245-... | lld:pubmed |
| pubmed-article:3133245 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:3133245 | pubmed:articleTitle | Ca2+ regulation of thyroid NADPH-dependent H2O2 generation. | lld:pubmed |
| pubmed-article:3133245 | pubmed:affiliation | Unité 96, INSERM, Unité de Recherche sur la Glande Thyroide et la Régulation Hormonale, Le Kremlin-Bicêtre, France. | lld:pubmed |
| pubmed-article:3133245 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3133245 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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