| pubmed-article:312893 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C2355612 | lld:lifeskim |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C0010583 | lld:lifeskim |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C0020522 | lld:lifeskim |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:312893 | lifeskim:mentions | umls-concept:C0205099 | lld:lifeskim |
| pubmed-article:312893 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:312893 | pubmed:dateCreated | 1979-9-1 | lld:pubmed |
| pubmed-article:312893 | pubmed:abstractText | Effect of treatment of mice with cyclophosphamide (CY) on the delayed hypersensitivity (DH) response was investigated in C57BL/6 mice. DH to methylated human serum albumin (MHSA) could be enhanced with CY in young mice but not in aged ones. DH enhancement with CY appeared to be due to elimination of suppressor T cells involved in DH. Effector T cells were also sensitive to CY, the damaging effect of CY on these latter cells was, however, transient suggesting the rapid recovery of effector T cells. The overshooting recovery of the effector T cells required the presence of the thymus. It is more probably that there are at least two distinct subpopulations of T cells in DH, effector T cells, and suppressor T cells. The distinction is already apparent in the thymus stage. The suppressor T cells, categorized as a central regulator, seem to be antigen nonspecific and regulate the more effectively the DH in young mice, thus physiological role of these cells in age-associated immune alterations is implicated. | lld:pubmed |
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| pubmed-article:312893 | pubmed:language | eng | lld:pubmed |
| pubmed-article:312893 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:312893 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:312893 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:312893 | pubmed:month | May | lld:pubmed |
| pubmed-article:312893 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:312893 | pubmed:author | pubmed-author:HaradaTT | lld:pubmed |
| pubmed-article:312893 | pubmed:author | pubmed-author:BabbLL | lld:pubmed |
| pubmed-article:312893 | pubmed:author | pubmed-author:MorikawaSS | lld:pubmed |
| pubmed-article:312893 | pubmed:author | pubmed-author:MitsuokaAA | lld:pubmed |
| pubmed-article:312893 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:312893 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:312893 | pubmed:volume | 149 | lld:pubmed |
| pubmed-article:312893 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:312893 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:312893 | pubmed:pagination | 1018-28 | lld:pubmed |
| pubmed-article:312893 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:312893 | pubmed:year | 1979 | lld:pubmed |
| pubmed-article:312893 | pubmed:articleTitle | Cyclophosphamide eliminates suppressor T cells in age-associated central regulation of delayed hypersensitivity in mice. | lld:pubmed |
| pubmed-article:312893 | pubmed:publicationType | Journal Article | lld:pubmed |
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