pubmed-article:3073091 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C2673643 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C0020621 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C0678226 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C0920563 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C1947976 | lld:lifeskim |
pubmed-article:3073091 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
pubmed-article:3073091 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:3073091 | pubmed:dateCreated | 1989-6-14 | lld:pubmed |
pubmed-article:3073091 | pubmed:abstractText | In a type II diabetic patient presenting with chronic hypokaliemia secondary to a selective renal tubulopathy, insulin sensitivity was explored three times during a euglycemic hyperinsulinemic clamp procedure at two rates of insulin infusion: 1 and 10 mU/kg/min: once before treatment of hypokaliemia and once after successful correction of hypokaliemia with indomethacine or spironolactone. During insulin infusion, a 20% dextrose solution was infused by a Biostator in order to maintain the patient's glycemia at 90 mg/dl. Amounts of glucose infused during the last 20 min of each 2 hour insulin infusion were (at 1 and 10 m/kg/min respectively): before treatment (K+ = 2.7 mmol/l): 2.4 and 8.4 mg/kg/min; after spironolactone (K+ = 3.9 mmol/l): 3.3 and 15.4 mg/kg/min; after indomethacine (K+ = 3.7 mmol/l): 5 and 19 mg/kg/min after stopping drugs (K+ = 2.9 mmol/l): 2.5 and 5.3 mg/kg/min. These data suggest that potassium metabolism plays a critical role in the mechanisms of insulin sensitivity. | lld:pubmed |
pubmed-article:3073091 | pubmed:language | fre | lld:pubmed |
pubmed-article:3073091 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3073091 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:3073091 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3073091 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3073091 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3073091 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:3073091 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:3073091 | pubmed:month | Dec | lld:pubmed |
pubmed-article:3073091 | pubmed:issn | 0338-1684 | lld:pubmed |
pubmed-article:3073091 | pubmed:author | pubmed-author:TourniaireJJ | lld:pubmed |
pubmed-article:3073091 | pubmed:author | pubmed-author:GarretLL | lld:pubmed |
pubmed-article:3073091 | pubmed:author | pubmed-author:BajardLL | lld:pubmed |
pubmed-article:3073091 | pubmed:author | pubmed-author:RebattuBB | lld:pubmed |
pubmed-article:3073091 | pubmed:author | pubmed-author:HarfouchMM | lld:pubmed |
pubmed-article:3073091 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:3073091 | pubmed:volume | 14 | lld:pubmed |
pubmed-article:3073091 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:3073091 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:3073091 | pubmed:pagination | 717-20 | lld:pubmed |
pubmed-article:3073091 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
pubmed-article:3073091 | pubmed:meshHeading | pubmed-meshheading:3073091-... | lld:pubmed |
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pubmed-article:3073091 | pubmed:year | 1988 | lld:pubmed |
pubmed-article:3073091 | pubmed:articleTitle | [Restoration of insulin sensitivity after correction of hypokalemia due to chronic tubulopathy in a diabetic patient]. | lld:pubmed |
pubmed-article:3073091 | pubmed:affiliation | Clinique Endocrinologique, Hôpital de l'Antiquaille, Lyon. | lld:pubmed |
pubmed-article:3073091 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:3073091 | pubmed:publicationType | English Abstract | lld:pubmed |
pubmed-article:3073091 | pubmed:publicationType | Case Reports | lld:pubmed |