| pubmed-article:3043226 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3043226 | lifeskim:mentions | umls-concept:C0524637 | lld:lifeskim |
| pubmed-article:3043226 | lifeskim:mentions | umls-concept:C0034790 | lld:lifeskim |
| pubmed-article:3043226 | lifeskim:mentions | umls-concept:C1335671 | lld:lifeskim |
| pubmed-article:3043226 | pubmed:issue | 6181 | lld:pubmed |
| pubmed-article:3043226 | pubmed:dateCreated | 1988-9-12 | lld:pubmed |
| pubmed-article:3043226 | pubmed:abstractText | The four distinct T-cell antigen receptor polypeptides (alpha, beta, gamma, delta) form two different heterodimers (alpha:beta and gamma:delta) that are very similar to immunoglobulins in primary sequence, gene organization and modes of rearrangement. Whereas antibodies have both soluble and membrane forms that can bind to antigens alone, T-cell receptors exist only on cell surfaces and recognize antigen fragments only when they are embedded in major histocompatibility complex (MHC) molecules. Patterns of diversity in T-cell receptor genes together with structural features of immunoglobulin and MHC molecules suggest a model for how this recognition might occur. This view of T-cell recognition has implications for how the receptors might be selected in the thymus and how they (and immunoglobulins) may have arisen during evolution. | lld:pubmed |
| pubmed-article:3043226 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3043226 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3043226 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3043226 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3043226 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3043226 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:3043226 | pubmed:issn | 0028-0836 | lld:pubmed |
| pubmed-article:3043226 | pubmed:author | pubmed-author:DavisM MMM | lld:pubmed |
| pubmed-article:3043226 | pubmed:author | pubmed-author:BjorkmanP JPJ | lld:pubmed |
| pubmed-article:3043226 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3043226 | pubmed:day | 4 | lld:pubmed |
| pubmed-article:3043226 | pubmed:volume | 334 | lld:pubmed |
| pubmed-article:3043226 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3043226 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3043226 | pubmed:pagination | 395-402 | lld:pubmed |
| pubmed-article:3043226 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
| pubmed-article:3043226 | pubmed:meshHeading | pubmed-meshheading:3043226-... | lld:pubmed |
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| pubmed-article:3043226 | pubmed:meshHeading | pubmed-meshheading:3043226-... | lld:pubmed |
| pubmed-article:3043226 | pubmed:year | 1988 | lld:pubmed |
| pubmed-article:3043226 | pubmed:articleTitle | T-cell antigen receptor genes and T-cell recognition. | lld:pubmed |
| pubmed-article:3043226 | pubmed:affiliation | Howard Hughes Medical Institute, Stanford University Medical School, California 94305-5402. | lld:pubmed |
| pubmed-article:3043226 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3043226 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3043226 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:3043226 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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