| pubmed-article:3029407 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3029407 | lifeskim:mentions | umls-concept:C0206679 | lld:lifeskim |
| pubmed-article:3029407 | lifeskim:mentions | umls-concept:C0010825 | lld:lifeskim |
| pubmed-article:3029407 | lifeskim:mentions | umls-concept:C0014644 | lld:lifeskim |
| pubmed-article:3029407 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:3029407 | pubmed:dateCreated | 1987-4-22 | lld:pubmed |
| pubmed-article:3029407 | pubmed:abstractText | Polyvalent rabbit antisera against herpes simplex virus type 1 and 2 (HSV-1 and HSV-2), cytomegalovirus (CMV), and Epstein-Barr virus (EBV), monospecific antisera against affinity-purified HSV-2 glycoproteins gB and gG, and a panel of monoclonal antibodies against HSV and EBV proteins were used to analyze cross-reactive molecules in cells infected with the four herpesviruses. A combination of immunoprecipitation and Western blotting with these reagents was used to determine that all four viruses coded for a glycoprotein that cross-reacted with HSV-1 gB. CMV coded for proteins that cross-reacted with HSV-2 gC, gD, and gE. Both CMV and EBV coded for proteins that cross-reacted with HSV-2 gG. Antigenic counterparts to the p45 nucleocapsid protein of HSV-2 were present in HSV-1 and CMV, and counterparts of the major DNA-binding protein and the ribonucleotide reductase of HSV-1 were present in all the viruses. The EBV virion glycoprotein gp85 was immunoprecipitated by antisera to HSV-1, HSV-2, and CMV. Antisera to CMV and EBV neutralized the infectivity of both HSV-1 and HSV-2 at high concentrations. This suggests that cross-reactivity between these four human herpesviruses may have pathogenic as well as evolutionary significance. | lld:pubmed |
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| pubmed-article:3029407 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:3029407 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:3029407 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3029407 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:3029407 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:3029407 | pubmed:author | pubmed-author:BalachandranN... | lld:pubmed |
| pubmed-article:3029407 | pubmed:author | pubmed-author:Hutt-Fletcher... | lld:pubmed |
| pubmed-article:3029407 | pubmed:author | pubmed-author:ObaD EDE | lld:pubmed |
| pubmed-article:3029407 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3029407 | pubmed:volume | 61 | lld:pubmed |
| pubmed-article:3029407 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3029407 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3029407 | pubmed:pagination | 1125-35 | lld:pubmed |
| pubmed-article:3029407 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3029407 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:3029407 | pubmed:articleTitle | Antigenic cross-reactions among herpes simplex virus types 1 and 2, Epstein-Barr virus, and cytomegalovirus. | lld:pubmed |
| pubmed-article:3029407 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3029407 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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