| pubmed-article:3016518 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C0026626 | lld:lifeskim |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C0023978 | lld:lifeskim |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C0039551 | lld:lifeskim |
| pubmed-article:3016518 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:3016518 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:3016518 | pubmed:dateCreated | 1986-9-22 | lld:pubmed |
| pubmed-article:3016518 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3016518 | pubmed:abstractText | We tested the ability of sequences in the long terminal repeat (LTR) of a mink cell focus-forming (MCF) murine leukemia virus to function as an enhancer in a cell-type-specific manner. In a stable transformation assay, the MCF or Akv LTR and the simian virus 40 enhancer had similar activities in murine fibroblasts. In contrast, the MCF LTR had a significantly greater activity in murine T lymphoid cells than did either the simian virus 40 enhancer or the Akv LTR. | lld:pubmed |
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| pubmed-article:3016518 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3016518 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3016518 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3016518 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3016518 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:3016518 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:3016518 | pubmed:author | pubmed-author:YoshimuraF... | lld:pubmed |
| pubmed-article:3016518 | pubmed:author | pubmed-author:DavisonBB | lld:pubmed |
| pubmed-article:3016518 | pubmed:author | pubmed-author:ChaffinKK | lld:pubmed |
| pubmed-article:3016518 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3016518 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:3016518 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3016518 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3016518 | pubmed:pagination | 2832-5 | lld:pubmed |
| pubmed-article:3016518 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3016518 | pubmed:year | 1985 | lld:pubmed |
| pubmed-article:3016518 | pubmed:articleTitle | Murine leukemia virus long terminal repeat sequences can enhance gene activity in a cell-type-specific manner. | lld:pubmed |
| pubmed-article:3016518 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3016518 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:3016518 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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