| pubmed-article:2980505 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0020255 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0348026 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0024485 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0237881 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0220901 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C0750502 | lld:lifeskim |
| pubmed-article:2980505 | lifeskim:mentions | umls-concept:C1552835 | lld:lifeskim |
| pubmed-article:2980505 | pubmed:dateCreated | 1992-5-28 | lld:pubmed |
| pubmed-article:2980505 | pubmed:abstractText | The presence of periventricular high signal lesions and ventricular signal void was tabulated and compared in three groups: patients with unshunted hydrocephalus (n = 24), cerebral atrophy (n = 14), and a subgroup of treated hydrocephalus subjects studied both pre- and post-shunting (n = 6). We found that T2 high signal lesions and ventricular signal void are common in both hydrocephalus and atrophy and are of no value in distinguishing between the two. The pre-treatment presence or absence of periventricular high signal lesions and signal void were of no use in predicting the clinical outcome of shunting in hydrocephalus. | lld:pubmed |
| pubmed-article:2980505 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2980505 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2980505 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2980505 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2980505 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2980505 | pubmed:issn | 0365-5954 | lld:pubmed |
| pubmed-article:2980505 | pubmed:author | pubmed-author:GeorgeA EAE | lld:pubmed |
| pubmed-article:2980505 | pubmed:author | pubmed-author:de LeonM JMJ | lld:pubmed |
| pubmed-article:2980505 | pubmed:author | pubmed-author:PintoR SRS | lld:pubmed |
| pubmed-article:2980505 | pubmed:author | pubmed-author:StollmanA LAL | lld:pubmed |
| pubmed-article:2980505 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2980505 | pubmed:volume | 369 | lld:pubmed |
| pubmed-article:2980505 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2980505 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2980505 | pubmed:pagination | 388-91 | lld:pubmed |
| pubmed-article:2980505 | pubmed:dateRevised | 2008-4-9 | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:meshHeading | pubmed-meshheading:2980505-... | lld:pubmed |
| pubmed-article:2980505 | pubmed:year | 1986 | lld:pubmed |
| pubmed-article:2980505 | pubmed:articleTitle | Periventricular high signal lesions and signal void on magnetic resonance imaging in hydrocephalus. Diagnostic and prognostic significance. | lld:pubmed |
| pubmed-article:2980505 | pubmed:affiliation | Department of Radiology (Neuroradiology), New York University Medical Center, NY. | lld:pubmed |
| pubmed-article:2980505 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2980505 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
