Linked Life Data

2965156

Source:http://linkedlifedata.com/resource/pubmed/id/2965156

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pubmed-article:2965156pubmed:abstractTextSynthesis and expression of cell surface carbohydrates appear to be involved in recognition events associated with tumor invasion and metastasis. Thus, the potential of murine sarcoma L-1 cells to form experimental lung metastases after i.v. injection was assessed after inhibiting tumor cell protein glycosylation with tunicamycin, swainsonine, bromoconduritol, or 1-desoxynojirimycin. Incubation of sarcoma L-1 cells with 0.5 microgram (or above) of these substances/ml medium for 20-24 h significantly inhibited lung colonization. Cytotoxic side effects or additional organ manifestations could not be found. Gas liquid chromatographic examinations of carbohydrates from treated L-1 cells indicated that sugar synthesis was evidently inhibited. These results suggest that specific glycan structures on tumor cells are required for expression of the metastatic phenotype.lld:pubmed
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pubmed-article:2965156pubmed:articleTitleGlycoprotein modifications of sarcoma L-1 tumor cells by tunicamycin, swainsonine, bromoconduritol or 1-desoxynojirimycin treatment inhibits their metastatic lung colonization in Balb/c-mice.lld:pubmed
pubmed-article:2965156pubmed:affiliationInstitute of Hygiene, University of Cologne, Federal Republic of Germany.lld:pubmed
pubmed-article:2965156pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:2965156pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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