Linked Life Data

2889861

Source:http://linkedlifedata.com/resource/pubmed/id/2889861

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pubmed-article:2889861pubmed:abstractTextThe effect of fructose on liver metabolism in patients with hereditary fructose intolerance (HFI) and in heterozygotes for HFI was studied by 31P magnetic resonance spectroscopy (31P-MRS). In patients with HFI (n = 5) ingestion of small amounts of fructose was followed by an increase in sugar phosphates and decrease in inorganic phosphate (Pi) in the liver that could be detected by 31P-MRS. 31P-MRS could be used to diagnose fructose intolerance and to monitor the patients' compliance with a fructose-restricted diet. In heterozygotes (n = 8) 50 g fructose given orally led to accumulation of sugar phosphates and depletion of Pi in the liver. Fructose also induced a larger increase in plasma urate in heterozygotes than in control subjects. The effect of fructose on liver Pi and plasma urate was most pronounced in heterozygotes with gout (n = 3). Heterozygosity for HFI may predispose to hyperuricaemia.lld:pubmed
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pubmed-article:2889861pubmed:articleTitleStudy of hereditary fructose intolerance by use of 31P magnetic resonance spectroscopy.lld:pubmed
pubmed-article:2889861pubmed:affiliationMRC Clinical Magnetic Resonance Facility, John Radcliffe Hospital, Oxford.lld:pubmed
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