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pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0014834 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0001268 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0005516 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0002085 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C0559956 | lld:lifeskim |
pubmed-article:2848016 | lifeskim:mentions | umls-concept:C1524063 | lld:lifeskim |
pubmed-article:2848016 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:2848016 | pubmed:dateCreated | 1989-1-5 | lld:pubmed |
pubmed-article:2848016 | pubmed:abstractText | We replaced the Escherichia coli lexA gene by a segment of DNA coding for resistance to spectinomycin and streptomycin. The use of this segment expands the range of selectable markers usable for allele replacement. The availability of this null lexA mutation will facilitate genetic analysis of lexA and the SOS regulon. | lld:pubmed |
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pubmed-article:2848016 | pubmed:language | eng | lld:pubmed |
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pubmed-article:2848016 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:2848016 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2848016 | pubmed:month | Dec | lld:pubmed |
pubmed-article:2848016 | pubmed:issn | 0021-9193 | lld:pubmed |
pubmed-article:2848016 | pubmed:author | pubmed-author:HillS ASA | lld:pubmed |
pubmed-article:2848016 | pubmed:author | pubmed-author:LittleJ WJW | lld:pubmed |
pubmed-article:2848016 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2848016 | pubmed:volume | 170 | lld:pubmed |
pubmed-article:2848016 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2848016 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2848016 | pubmed:pagination | 5913-5 | lld:pubmed |
pubmed-article:2848016 | pubmed:dateRevised | 2010-9-9 | lld:pubmed |
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pubmed-article:2848016 | pubmed:year | 1988 | lld:pubmed |
pubmed-article:2848016 | pubmed:articleTitle | Allele replacement in Escherichia coli by use of a selectable marker for resistance to spectinomycin: replacement of the lexA gene. | lld:pubmed |
pubmed-article:2848016 | pubmed:affiliation | Department of Biochemistry and Molecular Biology, University of Arizona,Tucson 85721. | lld:pubmed |
pubmed-article:2848016 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2848016 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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