| pubmed-article:2767683 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C0027651 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C0153594 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C1520354 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C0002085 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
| pubmed-article:2767683 | lifeskim:mentions | umls-concept:C1553568 | lld:lifeskim |
| pubmed-article:2767683 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:2767683 | pubmed:dateCreated | 1989-9-29 | lld:pubmed |
| pubmed-article:2767683 | pubmed:abstractText | Constitutional and tumor genotypes defined by polymorphic DNA markers were examined in 31 testicular cancer patients. Constitutional karyotypes were analyzed and clinical data presented. We analyzed 11 loci representing 8 chromosomes, including regions frequently deleted in other types of cancer. Loss of 3p or 11p sequences was detected in 8 of 28 heterozygotes (28%) and in 5 of 20 heterozygotes (25%). This gives a combined total loss of 40%. The other autosomal loci tested showed no loss or a loss of less than 10% of alleles. We suggest that this loss of heterozygosity for genetic material on chromosome 3p or on 11p is nonrandom and important in the development of a major subset of testicular neoplasms. | lld:pubmed |
| pubmed-article:2767683 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2767683 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2767683 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2767683 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2767683 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:2767683 | pubmed:issn | 0888-7543 | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:WhiteRR | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:NakamuraYY | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:FossåS DSD | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:BrøggerAA | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:StenwigA EAE | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:BørresenA LAL | lld:pubmed |
| pubmed-article:2767683 | pubmed:author | pubmed-author:LotheR ARA | lld:pubmed |
| pubmed-article:2767683 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2767683 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:2767683 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2767683 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2767683 | pubmed:pagination | 134-8 | lld:pubmed |
| pubmed-article:2767683 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:2767683 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2767683 | pubmed:articleTitle | Loss of 3p or 11p alleles is associated with testicular cancer tumors. | lld:pubmed |
| pubmed-article:2767683 | pubmed:affiliation | Department of Genetics, Norwegian Radium Hospital, Montebello, Oslo. | lld:pubmed |
| pubmed-article:2767683 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2767683 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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