pubmed-article:2714635 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C0034865 | lld:lifeskim |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C1516520 | lld:lifeskim |
pubmed-article:2714635 | lifeskim:mentions | umls-concept:C1522492 | lld:lifeskim |
pubmed-article:2714635 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:2714635 | pubmed:dateCreated | 1989-6-19 | lld:pubmed |
pubmed-article:2714635 | pubmed:abstractText | The role of recBC functions has been tested for three types of chromosomal recombination events: (1) recombination between direct repeats to generate a deletion, (2) recombination between a small circular fragment and the chromosome, and (3) recombination between inversely oriented repeats to form an inversion. Deletion formation by recombination between direct repeats, which does not require a fully reciprocal exchange, is independent of recBC function. Circle integration and inversion formation are both stimulated by the recBC function; these events require full reciprocality. The results suggest that half-reciprocal exchanges can occur without recBC, but recBC functions greatly stimulate completion of a fully reciprocal exchange. We propose that chromosomal recombination is a two-step process, and recBC functions are primarily required for the second step. | lld:pubmed |
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pubmed-article:2714635 | pubmed:language | eng | lld:pubmed |
pubmed-article:2714635 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2714635 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:2714635 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2714635 | pubmed:month | Mar | lld:pubmed |
pubmed-article:2714635 | pubmed:issn | 0016-6731 | lld:pubmed |
pubmed-article:2714635 | pubmed:author | pubmed-author:RothJ RJR | lld:pubmed |
pubmed-article:2714635 | pubmed:author | pubmed-author:MahanM JMJ | lld:pubmed |
pubmed-article:2714635 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2714635 | pubmed:volume | 121 | lld:pubmed |
pubmed-article:2714635 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2714635 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2714635 | pubmed:pagination | 433-43 | lld:pubmed |
pubmed-article:2714635 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:2714635 | pubmed:year | 1989 | lld:pubmed |
pubmed-article:2714635 | pubmed:articleTitle | Role of recBC function in formation of chromosomal rearrangements: a two-step model for recombination. | lld:pubmed |
pubmed-article:2714635 | pubmed:affiliation | Department of Biology, University of Utah, Salt Lake City 84112. | lld:pubmed |
pubmed-article:2714635 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2714635 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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