| pubmed-article:2644240 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C0014834 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C0014442 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C0035143 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C0017362 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C1510824 | lld:lifeskim |
| pubmed-article:2644240 | lifeskim:mentions | umls-concept:C2348867 | lld:lifeskim |
| pubmed-article:2644240 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:2644240 | pubmed:dateCreated | 1989-3-16 | lld:pubmed |
| pubmed-article:2644240 | pubmed:abstractText | In Escherichia coli anaerobic growth lowers the basal or induced levels of numerous enzymes associated with aerobic metabolism. Mutations in arcA (dye) at min 0 relieve this pleiotropic anaerobic repression and render the cell sensitive to the redox dye toluidine blue. In this study we identified a second pleiotropic control gene, arcB, at min 69.5. Mutations, including a deletion, in this gene also relieved the anaerobic repression and caused sensitivity to toluidine blue. Mutations in arcA or arcB did not significantly change the catabolite repression of the target phi(sdh-lacZ) operon, in which lacZ is fused to a structural gene for succinate dehydrogenase, nor did the mutations strikingly influence the pattern of excretion products during glucose fermentation. The presence of arcA+ in a multicopy plasmid restored anaerobic repression in arcB mutants, as indicated by the expression of phi(sdh-lacZ). The arcB product might be a sensor protein for the redox or energy state of the arc regulatory system. | lld:pubmed |
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| pubmed-article:2644240 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2644240 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2644240 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2644240 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:2644240 | pubmed:issn | 0021-9193 | lld:pubmed |
| pubmed-article:2644240 | pubmed:author | pubmed-author:LinE CEC | lld:pubmed |
| pubmed-article:2644240 | pubmed:author | pubmed-author:IuchiSS | lld:pubmed |
| pubmed-article:2644240 | pubmed:author | pubmed-author:CameronD CDC | lld:pubmed |
| pubmed-article:2644240 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2644240 | pubmed:volume | 171 | lld:pubmed |
| pubmed-article:2644240 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2644240 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2644240 | pubmed:pagination | 868-73 | lld:pubmed |
| pubmed-article:2644240 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2644240 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2644240 | pubmed:articleTitle | A second global regulator gene (arcB) mediating repression of enzymes in aerobic pathways of Escherichia coli. | lld:pubmed |
| pubmed-article:2644240 | pubmed:affiliation | Department of Microbiology and Molecular Genetics, Harvard Medical School, Boston, Massachusetts 02115. | lld:pubmed |
| pubmed-article:2644240 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2644240 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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