| pubmed-article:2558959 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2558959 | lifeskim:mentions | umls-concept:C0031705 | lld:lifeskim |
| pubmed-article:2558959 | lifeskim:mentions | umls-concept:C0013139 | lld:lifeskim |
| pubmed-article:2558959 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
| pubmed-article:2558959 | lifeskim:mentions | umls-concept:C1176299 | lld:lifeskim |
| pubmed-article:2558959 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:2558959 | pubmed:dateCreated | 1990-3-15 | lld:pubmed |
| pubmed-article:2558959 | pubmed:abstractText | Activity of the P family of transposable elements in Drosophila melanogaster is regulated primarily by a cellular condition known as P cytotype. It has been hypothesized that P cytotype depends on a P element-encoded repressor of transposition and excision. We provide evidence in support of this idea by showing that two modified P elements, each with lesions affecting the fourth transposase exon, mimic most of the P cytotype effects. These elements were identified by means of two sensitive assays capable of detecting repression by a single P element. One assay makes use of cytotype-dependent gene expression of certain P element insertion mutations at the singed bristle locus. The other measures suppression of transposase activity from the unusually stable genomic P element, delta 2-3(99B), that normally produces transposase in both germinal and somatic tissues. The P cytotype-like effects include suppression of snw germline hypermutability, snw somatic mosaicism, pupal lethality, and gonadal dysgenic sterility. Unlike P cytotype, however, there was no reciprocal cross effect in the inheritance of repression. | lld:pubmed |
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| pubmed-article:2558959 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2558959 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2558959 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2558959 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2558959 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2558959 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:2558959 | pubmed:author | pubmed-author:EngelsW RWR | lld:pubmed |
| pubmed-article:2558959 | pubmed:author | pubmed-author:RobertsonH... | lld:pubmed |
| pubmed-article:2558959 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2558959 | pubmed:volume | 123 | lld:pubmed |
| pubmed-article:2558959 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2558959 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2558959 | pubmed:pagination | 815-24 | lld:pubmed |
| pubmed-article:2558959 | pubmed:dateRevised | 2010-9-9 | lld:pubmed |
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| pubmed-article:2558959 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2558959 | pubmed:articleTitle | Modified P elements that mimic the P cytotype in Drosophila melanogaster. | lld:pubmed |
| pubmed-article:2558959 | pubmed:affiliation | Department of Entomology, University of Illinois, Urbana 61801. | lld:pubmed |
| pubmed-article:2558959 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2558959 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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