| pubmed-article:2473861 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2473861 | lifeskim:mentions | umls-concept:C0175677 | lld:lifeskim |
| pubmed-article:2473861 | lifeskim:mentions | umls-concept:C0022663 | lld:lifeskim |
| pubmed-article:2473861 | lifeskim:mentions | umls-concept:C1817908 | lld:lifeskim |
| pubmed-article:2473861 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:2473861 | pubmed:dateCreated | 1989-9-1 | lld:pubmed |
| pubmed-article:2473861 | pubmed:abstractText | A new model of experimental glomerulonephritis (GN) in which a cell-mediated (delayed-type) reaction predominated was established. Cationized TNP-BSA conjugates were planted in the kidney of rats by perfusion via the renal artery. Rats which had been sensitized with the TNP hapten 7 days previously revealed marked exudative and proliferative changes in their glomeruli, accompanied by transient proteinuria. A cellular influx was seen without deposition of any autologous antibody. The resulting data are compatible with the idea that cell-mediated immune reactions can participate in tissue damage in glomerular disease. | lld:pubmed |
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| pubmed-article:2473861 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2473861 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2473861 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2473861 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2473861 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:2473861 | pubmed:issn | 0009-9104 | lld:pubmed |
| pubmed-article:2473861 | pubmed:author | pubmed-author:ShimizuFF | lld:pubmed |
| pubmed-article:2473861 | pubmed:author | pubmed-author:MoriokaTT | lld:pubmed |
| pubmed-article:2473861 | pubmed:author | pubmed-author:OiteTT | lld:pubmed |
| pubmed-article:2473861 | pubmed:author | pubmed-author:KagamiSS | lld:pubmed |
| pubmed-article:2473861 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2473861 | pubmed:volume | 76 | lld:pubmed |
| pubmed-article:2473861 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2473861 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2473861 | pubmed:pagination | 463-8 | lld:pubmed |
| pubmed-article:2473861 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2473861 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2473861 | pubmed:articleTitle | Hapten-specific cellular immune response producing glomerular injury. | lld:pubmed |
| pubmed-article:2473861 | pubmed:affiliation | Department of Immunology, Niigata University School of Medicine, Japan. | lld:pubmed |
| pubmed-article:2473861 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2473861 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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