| pubmed-article:2467922 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2467922 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:2467922 | lifeskim:mentions | umls-concept:C0027950 | lld:lifeskim |
| pubmed-article:2467922 | lifeskim:mentions | umls-concept:C0079460 | lld:lifeskim |
| pubmed-article:2467922 | lifeskim:mentions | umls-concept:C1327414 | lld:lifeskim |
| pubmed-article:2467922 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:2467922 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:2467922 | pubmed:dateCreated | 1989-5-15 | lld:pubmed |
| pubmed-article:2467922 | pubmed:abstractText | Granulocyte-macrophage colony-stimulating factor (GM-CSF) is known as an inducer of proliferation and functional activation of myeloid cells. This study was carried out to characterize the effects of GM-CSF on polymorphonuclear leukocytes (PMN) more extensively. Using Northern blot analysis, we show that PMN are able to accumulate mRNAs for different cytokines, including tumor necrosis factor-alpha (TNF-alpha); G-CSF, and M-CSF, all of which are involved in inflammation and hematopoiesis. Biological assays and immunoassays demonstrate that PMN translate these mRNAs, except TNF-alpha, into secretory proteins. However, the expression of these cytokines is dependent on stimulation by exogenous signals, preferentially provided by the T cell-derived lymphokine GM-CSF. Stimulation of hematopoiesis and amplification of defense mechanisms after T cell activation thus might involve not only monocytes but also PMN, a cell type previously believed to be biosynthetically inactive. | lld:pubmed |
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| pubmed-article:2467922 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2467922 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:2467922 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2467922 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:2467922 | pubmed:issn | 0021-9738 | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:MertelsmannRR | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:HerrmannFF | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:RiedelDD | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:LindemannAA | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:Ziegler-Heitb... | lld:pubmed |
| pubmed-article:2467922 | pubmed:author | pubmed-author:OsterWW | lld:pubmed |
| pubmed-article:2467922 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2467922 | pubmed:volume | 83 | lld:pubmed |
| pubmed-article:2467922 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2467922 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2467922 | pubmed:pagination | 1308-12 | lld:pubmed |
| pubmed-article:2467922 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2467922 | pubmed:meshHeading | pubmed-meshheading:2467922-... | lld:pubmed |
| pubmed-article:2467922 | pubmed:year | 1989 | lld:pubmed |
| pubmed-article:2467922 | pubmed:articleTitle | Granulocyte-macrophage colony-stimulating factor induces cytokine secretion by human polymorphonuclear leukocytes. | lld:pubmed |
| pubmed-article:2467922 | pubmed:affiliation | Department of Hematology, University of Mainz, Federal Republic of Germany. | lld:pubmed |
| pubmed-article:2467922 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2467922 | pubmed:publicationType | Retracted Publication | lld:pubmed |
| pubmed-article:2467922 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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