| pubmed-article:2441919 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2441919 | lifeskim:mentions | umls-concept:C0011860 | lld:lifeskim |
| pubmed-article:2441919 | lifeskim:mentions | umls-concept:C0014792 | lld:lifeskim |
| pubmed-article:2441919 | lifeskim:mentions | umls-concept:C0949653 | lld:lifeskim |
| pubmed-article:2441919 | lifeskim:mentions | umls-concept:C0205307 | lld:lifeskim |
| pubmed-article:2441919 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:2441919 | pubmed:dateCreated | 1987-9-30 | lld:pubmed |
| pubmed-article:2441919 | pubmed:abstractText | The diminished insulin secretion of type 2 diabetes might result from abnormal regulation of the potassium permeability which leads to beta-cell depolarization. The possibility of a generalized defect has been investigated in vitro by the stimulation of 86Rb efflux from red cells of type 2 diabetic patients by calcium ionophore and its inhibition by quinine. Diabetic subjects and control subjects had identical 86Rb efflux stimulated by 0.2-0.6 microM calcium ionophore A23187 and identical inhibition by quinine with mean Ki 6 microM and 4 microM quinine respectively for 0.2 microM ionophore and mean Ki 38 microM and 37 microM quinine respectively for 0.6 microM ionophore. | lld:pubmed |
| pubmed-article:2441919 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2441919 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2441919 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2441919 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2441919 | pubmed:month | May | lld:pubmed |
| pubmed-article:2441919 | pubmed:issn | 0265-5985 | lld:pubmed |
| pubmed-article:2441919 | pubmed:author | pubmed-author:Del VecchioMM | lld:pubmed |
| pubmed-article:2441919 | pubmed:author | pubmed-author:TurnerR CRC | lld:pubmed |
| pubmed-article:2441919 | pubmed:author | pubmed-author:O'RahillySS | lld:pubmed |
| pubmed-article:2441919 | pubmed:author | pubmed-author:BownEE | lld:pubmed |
| pubmed-article:2441919 | pubmed:author | pubmed-author:BurnettM AMA | lld:pubmed |
| pubmed-article:2441919 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2441919 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:2441919 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2441919 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2441919 | pubmed:pagination | 19-21 | lld:pubmed |
| pubmed-article:2441919 | pubmed:dateRevised | 2007-9-11 | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:meshHeading | pubmed-meshheading:2441919-... | lld:pubmed |
| pubmed-article:2441919 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:2441919 | pubmed:articleTitle | Normal calcium-activated potassium channel in red cells in type 2 diabetes. | lld:pubmed |
| pubmed-article:2441919 | pubmed:publicationType | Journal Article | lld:pubmed |
