| pubmed-article:2386087 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C1280551 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0007770 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0038525 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0017911 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0001480 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0205164 | lld:lifeskim |
| pubmed-article:2386087 | lifeskim:mentions | umls-concept:C0456205 | lld:lifeskim |
| pubmed-article:2386087 | pubmed:issue | 1-2 | lld:pubmed |
| pubmed-article:2386087 | pubmed:dateCreated | 1990-9-17 | lld:pubmed |
| pubmed-article:2386087 | pubmed:abstractText | The contents of glycogen and ATP in the major cerebral arteries were examined in dogs undergoing subarachnoid haemorrhage (SAH). SAH was produced by a single injection of autologous arterial blood (1 ml/kg body weight) into the cisterna magna. Vertebral angiograms showed biphasic basilar arterial narrowings after the injection of blood: Early arterial narrowing occurred immediately after the injection and continued for a few hours. Late arterial narrowing occurred from Day 1 to Day 14 of post-SAH period, and recovered to the normal level on Day 21 of post-SAH period. The content of glycogen in the large pial arteries significantly decreased from Day 1 to Day 14 and returned to the control level on Day 21. The content of ATP in the large pial arteries also decreased from Day 1 to Day 7 and returned to the control level on Day 14. These results show that energy stores in the major cerebral arteries might be diminished during late arterial narrowing. | lld:pubmed |
| pubmed-article:2386087 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2386087 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2386087 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2386087 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2386087 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2386087 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2386087 | pubmed:issn | 0001-6268 | lld:pubmed |
| pubmed-article:2386087 | pubmed:author | pubmed-author:OkamotoSS | lld:pubmed |
| pubmed-article:2386087 | pubmed:author | pubmed-author:KikuchiHH | lld:pubmed |
| pubmed-article:2386087 | pubmed:author | pubmed-author:UemuraYY | lld:pubmed |
| pubmed-article:2386087 | pubmed:author | pubmed-author:NozakiKK | lld:pubmed |
| pubmed-article:2386087 | pubmed:author | pubmed-author:YanamotoHH | lld:pubmed |
| pubmed-article:2386087 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2386087 | pubmed:volume | 104 | lld:pubmed |
| pubmed-article:2386087 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2386087 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2386087 | pubmed:pagination | 38-41 | lld:pubmed |
| pubmed-article:2386087 | pubmed:dateRevised | 2009-11-11 | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:meshHeading | pubmed-meshheading:2386087-... | lld:pubmed |
| pubmed-article:2386087 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2386087 | pubmed:articleTitle | Changes of glycogen and ATP contents of the major cerebral arteries after experimentally produced subarachnoid haemorrhage in the dog. | lld:pubmed |
| pubmed-article:2386087 | pubmed:affiliation | Department of Neurosurgery, Faculty of Medicine, Kyoto University, Japan. | lld:pubmed |
| pubmed-article:2386087 | pubmed:publicationType | Journal Article | lld:pubmed |
