| pubmed-article:2242698 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2242698 | lifeskim:mentions | umls-concept:C0035648 | lld:lifeskim |
| pubmed-article:2242698 | lifeskim:mentions | umls-concept:C0009324 | lld:lifeskim |
| pubmed-article:2242698 | lifeskim:mentions | umls-concept:C0596244 | lld:lifeskim |
| pubmed-article:2242698 | lifeskim:mentions | umls-concept:C0333348 | lld:lifeskim |
| pubmed-article:2242698 | lifeskim:mentions | umls-concept:C1384115 | lld:lifeskim |
| pubmed-article:2242698 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:2242698 | pubmed:dateCreated | 1991-1-3 | lld:pubmed |
| pubmed-article:2242698 | pubmed:abstractText | Elevated levels of luminal nitrite and a lowered luminal pH were found in 77 percent of patients with acute ulcerative colitis. No luminal nitrite was found in healthy control subjects. Nitrites are a secretory product of activated macrophages and neutrophils of the lamina propria, whereas the lowered luminal pH is due to diminished bicarbonate formation by impaired colonocytes. A hypothesis is put forward that nitrites, lowered pH, and bacterial amines are conducive to formation of carcinogenic n-nitroso compounds, which reflect a cancer risk in patients with ulcerative colitis dependent on the type and extent of inflammatory cell activation as well as metabolic impairment of colonic epithelial cells. | lld:pubmed |
| pubmed-article:2242698 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2242698 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2242698 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2242698 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2242698 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2242698 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2242698 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:2242698 | pubmed:issn | 0012-3706 | lld:pubmed |
| pubmed-article:2242698 | pubmed:author | pubmed-author:RoedigerW EWE | lld:pubmed |
| pubmed-article:2242698 | pubmed:author | pubmed-author:RadcliffeB... | lld:pubmed |
| pubmed-article:2242698 | pubmed:author | pubmed-author:LawsonM JMJ | lld:pubmed |
| pubmed-article:2242698 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2242698 | pubmed:volume | 33 | lld:pubmed |
| pubmed-article:2242698 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2242698 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2242698 | pubmed:pagination | 1034-6 | lld:pubmed |
| pubmed-article:2242698 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:2242698 | pubmed:meshHeading | pubmed-meshheading:2242698-... | lld:pubmed |
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| pubmed-article:2242698 | pubmed:meshHeading | pubmed-meshheading:2242698-... | lld:pubmed |
| pubmed-article:2242698 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2242698 | pubmed:articleTitle | Nitrite from inflammatory cells--a cancer risk factor in ulcerative colitis? | lld:pubmed |
| pubmed-article:2242698 | pubmed:affiliation | Department of Surgery, University of Adelaide, Australia. | lld:pubmed |
| pubmed-article:2242698 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2242698 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:2242698 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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