pubmed-article:21856480 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C0949656 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C1293122 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C0039155 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C1512045 | lld:lifeskim |
pubmed-article:21856480 | lifeskim:mentions | umls-concept:C2932035 | lld:lifeskim |
pubmed-article:21856480 | pubmed:issue | 9792 | lld:pubmed |
pubmed-article:21856480 | pubmed:dateCreated | 2011-8-22 | lld:pubmed |
pubmed-article:21856480 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:abstractText | Decreased systolic function is central to the pathogenesis of heart failure in millions of patients worldwide, but mechanism-related adverse effects restrict existing inotropic treatments. This study tested the hypothesis that omecamtiv mecarbil, a selective cardiac myosin activator, will augment cardiac function in human beings. | lld:pubmed |
pubmed-article:21856480 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:language | eng | lld:pubmed |
pubmed-article:21856480 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:21856480 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21856480 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21856480 | pubmed:month | Aug | lld:pubmed |
pubmed-article:21856480 | pubmed:issn | 1474-547X | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:SchillerNelso... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:TeerlinkJohn... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:WolffAndrew... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:GoldmanJonath... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:HabibzadehMoh... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:SaikaliKhalil... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:ClarkeCyril... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:MalikFady IFI | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:BeeRachelR | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:LeeJacqueline... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:ChenMichael... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:EscandonRafae... | lld:pubmed |
pubmed-article:21856480 | pubmed:author | pubmed-author:ElliottLyndse... | lld:pubmed |
pubmed-article:21856480 | pubmed:copyrightInfo | Copyright © 2011 Elsevier Ltd. All rights reserved. | lld:pubmed |
pubmed-article:21856480 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21856480 | pubmed:day | 20 | lld:pubmed |
pubmed-article:21856480 | pubmed:volume | 378 | lld:pubmed |
pubmed-article:21856480 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21856480 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21856480 | pubmed:pagination | 667-75 | lld:pubmed |
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pubmed-article:21856480 | pubmed:meshHeading | pubmed-meshheading:21856480... | lld:pubmed |
pubmed-article:21856480 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21856480 | pubmed:articleTitle | Dose-dependent augmentation of cardiac systolic function with the selective cardiac myosin activator, omecamtiv mecarbil: a first-in-man study. | lld:pubmed |
pubmed-article:21856480 | pubmed:affiliation | Section of Cardiology, San Francisco Veterans Affairs Medical Center, University of California, San Francisco, CA 94121-1545, USA. john.teerlink@ucsf.edu | lld:pubmed |
pubmed-article:21856480 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21856480 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
pubmed-article:21856480 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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