| pubmed-article:21620866 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0015259 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0041904 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0220931 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0011847 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C1540067 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0311404 | lld:lifeskim |
| pubmed-article:21620866 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:21620866 | pubmed:issue | 1-2 | lld:pubmed |
| pubmed-article:21620866 | pubmed:dateCreated | 2011-6-21 | lld:pubmed |
| pubmed-article:21620866 | pubmed:abstractText | MuRF1 E3 ubiquitin ligase has been identified as a mediator of skeletal muscle wasting in various skeletal muscle atrophy models, and its expression is upregulated by oxidative stress. Exercise training could decrease oxidative stress and restore the atrophied skeletal muscle. Here, our aim was to investigate whether exercise training has any effect on MuRF1 expression in rats with diabetes. | lld:pubmed |
| pubmed-article:21620866 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21620866 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21620866 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21620866 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21620866 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21620866 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21620866 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21620866 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21620866 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:21620866 | pubmed:issn | 1879-0631 | lld:pubmed |
| pubmed-article:21620866 | pubmed:author | pubmed-author:WangSongS | lld:pubmed |
| pubmed-article:21620866 | pubmed:author | pubmed-author:ZhaoZhi-WeiZW | lld:pubmed |
| pubmed-article:21620866 | pubmed:author | pubmed-author:ChenGuo-QingG... | lld:pubmed |
| pubmed-article:21620866 | pubmed:author | pubmed-author:YangYue-QinYQ | lld:pubmed |
| pubmed-article:21620866 | pubmed:author | pubmed-author:MouCai-YingCY | lld:pubmed |
| pubmed-article:21620866 | pubmed:copyrightInfo | Copyright © 2011 Elsevier Inc. All rights reserved. | lld:pubmed |
| pubmed-article:21620866 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21620866 | pubmed:day | 4 | lld:pubmed |
| pubmed-article:21620866 | pubmed:volume | 89 | lld:pubmed |
| pubmed-article:21620866 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21620866 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21620866 | pubmed:pagination | 44-9 | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:meshHeading | pubmed-meshheading:21620866... | lld:pubmed |
| pubmed-article:21620866 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21620866 | pubmed:articleTitle | Exercise training has beneficial anti-atrophy effects by inhibiting oxidative stress-induced MuRF1 upregulation in rats with diabetes. | lld:pubmed |
| pubmed-article:21620866 | pubmed:affiliation | Department of Health Science, Wuhan Institute of Physical Education, Luoyu Road No. 461, Wuhan City, Hubei Province, China. | lld:pubmed |
| pubmed-article:21620866 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21620866 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:21620866 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:140939 | entrezgene:pubmed | pubmed-article:21620866 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:21620866 | lld:entrezgene |
