| pubmed-article:21562757 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0069676 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C2711227 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0017725 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:21562757 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:21562757 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:21562757 | pubmed:dateCreated | 2011-7-8 | lld:pubmed |
| pubmed-article:21562757 | pubmed:abstractText | Obesity is strongly associated with the development of non-alcoholic fatty liver disease (NAFLD). The cytokine osteopontin (OPN) was recently shown to be involved in obesity-induced adipose tissue inflammation and reduced insulin response. Accumulating evidence links OPN to the pathogenesis of NAFLD. Here we aimed to identify the role of OPN in obesity-associated hepatic steatosis and impaired hepatic glucose metabolism. | lld:pubmed |
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| pubmed-article:21562757 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21562757 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21562757 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21562757 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21562757 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21562757 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21562757 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21562757 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:21562757 | pubmed:issn | 1432-0428 | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:KahleMM | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:PfauBB | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:MairMM | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:PlutzkyJJ | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:KenneyEE | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:Hrabé de... | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:StulnigT MTM | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:NeschenSS | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:ZeydaMM | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:KieferF WFW | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:NeuhoferAA | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:LegererBB | lld:pubmed |
| pubmed-article:21562757 | pubmed:author | pubmed-author:SchledererMM | lld:pubmed |
| pubmed-article:21562757 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21562757 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:21562757 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21562757 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21562757 | pubmed:pagination | 2132-42 | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:meshHeading | pubmed-meshheading:21562757... | lld:pubmed |
| pubmed-article:21562757 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21562757 | pubmed:articleTitle | Osteopontin deficiency protects against obesity-induced hepatic steatosis and attenuates glucose production in mice. | lld:pubmed |
