| pubmed-article:21537467 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21537467 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:21537467 | lifeskim:mentions | umls-concept:C0023820 | lld:lifeskim |
| pubmed-article:21537467 | lifeskim:mentions | umls-concept:C0441472 | lld:lifeskim |
| pubmed-article:21537467 | lifeskim:mentions | umls-concept:C0074992 | lld:lifeskim |
| pubmed-article:21537467 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:21537467 | pubmed:dateCreated | 2011-5-3 | lld:pubmed |
| pubmed-article:21537467 | pubmed:abstractText | The reverse cholesterol transport mediated by high-density lipoprotein (HDL) is an important mechanism for maintaining body cholesterol, and hence, the crucial anti-atherogenic action of the lipoprotein. Recent studies, however, have shown that HDL exerts a variety of anti-inflammatory and anti-atherogenic actions independently of cholesterol metabolism. The present review provides an overview of the roles of sphingosine 1-phosphate (S1P)/S1P receptor and apolipoprotein A-I/scavenger receptor class B type I systems in the anti-atherogenic HDL actions. In addition, the physiological significance of the existence of S1P in the HDL particles is discussed. | lld:pubmed |
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