pubmed-article:21536970 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:21536970 | lifeskim:mentions | umls-concept:C0521425 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C1414627 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C0449560 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C0205460 | lld:lifeskim |
pubmed-article:21536970 | lifeskim:mentions | umls-concept:C0332120 | lld:lifeskim |
pubmed-article:21536970 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:21536970 | pubmed:dateCreated | 2011-9-6 | lld:pubmed |
pubmed-article:21536970 | pubmed:abstractText | Polymorphisms in the gene encoding the glucocorticoid receptor (GR) regulating co-chaperone FKBP5 have been shown to alter GR sensitivity and are associated with an increased risk to develop posttraumatic stress disorder (PTSD). | lld:pubmed |
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pubmed-article:21536970 | pubmed:language | eng | lld:pubmed |
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pubmed-article:21536970 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:21536970 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21536970 | pubmed:month | Sep | lld:pubmed |
pubmed-article:21536970 | pubmed:issn | 1538-3636 | lld:pubmed |
pubmed-article:21536970 | pubmed:author | pubmed-author:HolsboerFlori... | lld:pubmed |
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pubmed-article:21536970 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21536970 | pubmed:volume | 68 | lld:pubmed |
pubmed-article:21536970 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21536970 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21536970 | pubmed:pagination | 901-10 | lld:pubmed |
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pubmed-article:21536970 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21536970 | pubmed:articleTitle | Using polymorphisms in FKBP5 to define biologically distinct subtypes of posttraumatic stress disorder: evidence from endocrine and gene expression studies. | lld:pubmed |
pubmed-article:21536970 | pubmed:affiliation | Max Planck Institute of Psychiatry, Kraepelinstrasse, Munich, Germany. | lld:pubmed |
pubmed-article:21536970 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21536970 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:21536970 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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