pubmed-article:2152250 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2152250 | lifeskim:mentions | umls-concept:C0023896 | lld:lifeskim |
pubmed-article:2152250 | lifeskim:mentions | umls-concept:C0677043 | lld:lifeskim |
pubmed-article:2152250 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:2152250 | lifeskim:mentions | umls-concept:C0042469 | lld:lifeskim |
pubmed-article:2152250 | lifeskim:mentions | umls-concept:C0750729 | lld:lifeskim |
pubmed-article:2152250 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:2152250 | pubmed:dateCreated | 1993-1-7 | lld:pubmed |
pubmed-article:2152250 | pubmed:abstractText | 24 patients with alcoholic intake were classified according to the amount of alcohol ingestion; clinical symptoms and signs, liver function tests (bilirubin, aminotransferases and prothrombin time) were analyzed. In all patients a percutaneous liver biopsy was performed and tissue stained by hematoxylin-eosin, wilder reticulin and Mallory trichromic. 9 Histologic criteria were analyzed. 4 groups according to the histology were identified. Group 1 (5 patients) hepatic fibrosis and/or fatty liver. Group 2 (5 patients) alcoholic hepatitis. Group 3 (10 patients) cirrhosis. Group 4 (4 patients) normal. 20% of patients with fatty liver, 80% of alcoholic hepatitis and 100% of cirrhotics referred ingestion or more than 160 g of ethanol and important correlation between liver histological damage and alcohol ingestion. Telangiectasia was the most common clinical finding and present in all hepatitis, 70% of cirrhotics and only 20% of fatty livers. Hemosiderosis was found in 60% of cirrhotics and in alcoholic hepatitis. Only 40% of patients with fatty liver and inflammatory cells while this was evident in all patients with alcoholic hepatitis and those with cirrhosis. Mallory bodies were identified in only 20% of cirrhotics and in none of the alcoholic hepatitis. The results suggest that there are significant differences from a histological and clinical point of view that distinguish alcoholic liver disease as seen in Venezuela from that reported in other countries. | lld:pubmed |
pubmed-article:2152250 | pubmed:language | spa | lld:pubmed |
pubmed-article:2152250 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2152250 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:2152250 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2152250 | pubmed:issn | 0016-3503 | lld:pubmed |
pubmed-article:2152250 | pubmed:author | pubmed-author:Pérez... | lld:pubmed |
pubmed-article:2152250 | pubmed:author | pubmed-author:HamannCC | lld:pubmed |
pubmed-article:2152250 | pubmed:author | pubmed-author:ArochaRR | lld:pubmed |
pubmed-article:2152250 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2152250 | pubmed:volume | 44 | lld:pubmed |
pubmed-article:2152250 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2152250 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2152250 | pubmed:pagination | 15-20 | lld:pubmed |
pubmed-article:2152250 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:2152250 | pubmed:articleTitle | [Alcoholic liver disease in Venezuela. Clinical hepato-functional and histopathologic course]. | lld:pubmed |
pubmed-article:2152250 | pubmed:affiliation | Servicio de Gastroenterología, Hospital Dr. Ildemaro Salas, IVSS, Caracas. | lld:pubmed |
pubmed-article:2152250 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2152250 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:2152250 | pubmed:publicationType | English Abstract | lld:pubmed |