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pubmed-article:21481001pubmed:abstractTextCleft palate is a frequent congenital malformation with a heterogeneous etiology, for which folic acid (FA) supplementation has a protective effect. To gain more insight into the molecular pathways affected by FA, TGF-? signaling and apoptosis in mouse embryonic palatal mesenchymal (MEPM) cells of all-trans retinoic acid (ATRA)-induced cleft palate in organ culture were tested.lld:pubmed
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pubmed-article:21481001pubmed:authorpubmed-author:ChenDanDlld:pubmed
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pubmed-article:21481001pubmed:authorpubmed-author:LiuZhonghuaZlld:pubmed
pubmed-article:21481001pubmed:authorpubmed-author:WangAnxunAlld:pubmed
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pubmed-article:21481001pubmed:authorpubmed-author:DingXueqiangXlld:pubmed
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pubmed-article:21481001pubmed:authorpubmed-author:HeQiantingQlld:pubmed
pubmed-article:21481001pubmed:copyrightInfo© 2010 John Wiley & Sons A/S.lld:pubmed
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pubmed-article:21481001pubmed:volume40lld:pubmed
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pubmed-article:21481001pubmed:year2011lld:pubmed
pubmed-article:21481001pubmed:articleTitleFolic acid rescue of ATRA-induced cleft palate by restoring the TGF-? signal and inhibiting apoptosis.lld:pubmed
pubmed-article:21481001pubmed:affiliationDepartment of Oral and Maxillofacial surgery, the First Affiliated Hospital, Sun Yat-sen University, Guangzhou, Guangdong, China.lld:pubmed
pubmed-article:21481001pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:21481001pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed