| pubmed-article:21471723 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0205054 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0521451 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0132555 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C1999216 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:21471723 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:21471723 | pubmed:issue | 3-4 | lld:pubmed |
| pubmed-article:21471723 | pubmed:dateCreated | 2011-4-7 | lld:pubmed |
| pubmed-article:21471723 | pubmed:abstractText | Mitochondrial dysfunction plays important roles in the development of diabetes. Elevated nitric oxide (NO) synthase inhibitor asymmetric dimethylarginine (ADMA) has been shown to be closely related to diabetes. But the relationship between them in diabetes has not been determined. This study was to explore the role of ADMA in hepatic mitochondrial dysfunction and its potential mechanisms in diabetic rats and hepatocytes. | lld:pubmed |
| pubmed-article:21471723 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21471723 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21471723 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21471723 | pubmed:issn | 1421-9778 | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:LuoJian-DongJ... | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:ErtlI OIO | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:XiongYanY | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:ChenMin-Sheng... | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:XuWen-JuanWJ | lld:pubmed |
| pubmed-article:21471723 | pubmed:author | pubmed-author:LengYi-PingYP | lld:pubmed |
| pubmed-article:21471723 | pubmed:copyrightInfo | Copyright © 2011 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:21471723 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21471723 | pubmed:volume | 27 | lld:pubmed |
| pubmed-article:21471723 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21471723 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21471723 | pubmed:pagination | 341-52 | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
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| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:meshHeading | pubmed-meshheading:21471723... | lld:pubmed |
| pubmed-article:21471723 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21471723 | pubmed:articleTitle | Contribution of endogenous inhibitor of nitric oxide synthase to hepatic mitochondrial dysfunction in streptozotocin-induced diabetic rats. | lld:pubmed |
| pubmed-article:21471723 | pubmed:affiliation | Department of Pharmacology, Guangzhou Medical University, Guangzhou, PR China. | lld:pubmed |
| pubmed-article:21471723 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21471723 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
