Phosphomimetic substitution of heterogeneous nuclear ribonucleoprotein A1 at serine 199 abolishes AKT-dependent internal ribosome entry site-transacting factor (ITAF) function via effects on strand annealing and results in mammalian target of rapamycin complex 1 (mTORC1) inhibitor sensitivity.
Source:http://linkedlifedata.com/resource/pubmed/id/21454539
J. Biol. Chem.
2011
May
6
286
18
16402-13
