| pubmed-article:21437208 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21437208 | lifeskim:mentions | umls-concept:C1332408 | lld:lifeskim |
| pubmed-article:21437208 | lifeskim:mentions | umls-concept:C0228174 | lld:lifeskim |
| pubmed-article:21437208 | lifeskim:mentions | umls-concept:C0042396 | lld:lifeskim |
| pubmed-article:21437208 | lifeskim:mentions | umls-concept:C0039082 | lld:lifeskim |
| pubmed-article:21437208 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:21437208 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:21437208 | pubmed:dateCreated | 2011-3-25 | lld:pubmed |
| pubmed-article:21437208 | pubmed:abstractText | Reversible cerebral vasoconstriction syndrome (RCVS) could be complicated by cerebral ischemic events. Hypothetical mechanisms of RCVS involve endothelial dysfunction and sympathetic overactivity, both of which were reported to be related to brain-derived neurotrophic factor (BDNF). The study investigated the association between functional BDNF Val66Met polymorphism and RCVS. | lld:pubmed |
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| pubmed-article:21437208 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:21437208 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:21437208 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21437208 | pubmed:issn | 1932-6203 | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:FuhJong-LingJ... | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:HongChen-JeeC... | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:TsaiShih-JenS... | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:WangShuu-Jiun... | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:ChenShih-PinS... | lld:pubmed |
| pubmed-article:21437208 | pubmed:author | pubmed-author:YangAlbert... | lld:pubmed |
| pubmed-article:21437208 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21437208 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:21437208 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21437208 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21437208 | pubmed:pagination | e18024 | lld:pubmed |
| pubmed-article:21437208 | pubmed:dateRevised | 2011-7-27 | lld:pubmed |
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| pubmed-article:21437208 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21437208 | pubmed:articleTitle | Brain-derived neurotrophic factor gene Val66Met polymorphism modulates reversible cerebral vasoconstriction syndromes. | lld:pubmed |
| pubmed-article:21437208 | pubmed:affiliation | Department of Neurology, Neurological Institute, Taipei Veterans General Hospital, Taipei, Taiwan. | lld:pubmed |
| pubmed-article:21437208 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21437208 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:627 | entrezgene:pubmed | pubmed-article:21437208 | lld:entrezgene |
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